Index of Online Supplemental Material for
J. Cell Biol. 10.1083/jcb.200701006
Grande-Garcia et al.

• Figure S1 Absence of Cav1 does not delay cell spreading.

• Figure S2 Constitutively active Rho and dominant-negative mutants of Rac and Cdc42 restore normal polarity in Cav1^-/- fibroblasts.

• Figure S3 A dominant-negative p190RhoGAP construct rescues polarity in Cav1^-/- fibroblasts.

• Figure S4 Src inhibition and p190RhoGAP knockdown in Cav1^-/- MEFs restore the WT morphology pattern.

• Figure S5 Total and tyrosine phosphorylated p190RhoGAP are elevated in Cav1^-/- MEFs.

• Video 1 Adhesion turnover in WT MEFs.

• Video 2 Adhesion turnover in Cav1^-/- MEFS.

• Video 3 Protrusion-retraction analysis of WT MEFs.

• Video 4 Protrusion-retraction analysis of Cav1^-/- MEFs.

• Video 5 Random migration of WT MEFs.

• Video 6 Random migration of Cav1^-/- MEFs.

• Video 7 Random migration of Cav1^-/- MEFs reconstituted with Cav1.

• Video 8 In vitro wound-healing assays in WT MEFs.

• Video 9 In vitro wound-healing assay in Cav1^-/- MEFs.

• Video 10 In vitro wound-healing assay in Cav1^-/- MEFs reconstituted with Cav1.