Ectopic Expression of Human MutS Homologue 2 on Renal Carcinoma Cells Is Induced by Oxidative Stress with Interleukin-18 Promotion via p38 Mitogen-activated Protein Kinase (MAPK) and c-Jun N-terminal Kinase (JNK) Signaling Pathways

Supplemental Data

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• suppl Fig 1 and Fig 2 (.pdf, 302 KB) - Supplemental Fig. 1. Regulatory function of the MEK/ERK pathway in ectopic expression of hMSH2 during oxidative stress. Supplemental Fig. 2. Surface expression of hMSH2 on GO-stimulated G401 cells after treatment with ATF3 siRNAs for 24 or 36h.