Inflammatory Monocyte Recruitment Is Regulated by Interleukin-23 during Systemic Bacterial Infection

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  Fig. S1. Treatment with the anti-Ly6G antibody depletes neutrophils from the spleen, liver, and blood. Fig. S2. Optimal monocyte recruitment to the spleen during a low-dose L. monocytogenes infection requires IL-23. Fig. S3. IL-23 does not regulate the production of CCL2 under homeostatic conditions.

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