Dynamic Interaction of Stress Granules, DDX3X, and IKK-α Mediates Multiple Functions in Hepatitis C Virus Infection

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  Fig. S1 (HCV 3′ UTR RNA- or HCV-induced DDX3X–IKK-α granules do not colocalize with mitochondria, peroxisomes, or autophagosomes.)

  Fig. S2 (HCV-induced DDX3X–IKK-α–G3BP1 granules colocalize with SG and Pbody markers.)

  Fig. S3 (Dynamic association of DDX3X with HCV core protein and LDs during HCV infection.)

  Fig. S4 (Dynamic redistribution of SGs towards LDs during HCV infection.)

  Fig. S5 (SGs are not induced in the absence of infectious HCV.)

  Fig. S6 (Effects of DDX3X or SG protein silencing in cells on productive HCV infection.)

  Fig. S7 (Knockdown of various SG-associated proteins exhibited different effects on SG formation and DDX3X–IKK-α aggregation.)

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