Human Endometrial Fibroblasts Derived from Mesenchymal Progenitors Inherit Progesterone Resistance and Acquire an Inflammatory Phenotype in the Endometrial Niche in Endometriosis

Supplemental Data

Supplemental Tables S1-S7.

  • Supplemental Table S1 - Subject characteristics.
  • Supplemental Table S2A - Differential gene expression comparisons among different endometrial cell types.
  • Supplemental Table S2B - Differential gene expression among FACS-isolated and cultured eMSC and eSF from control and endometriosis.
  • Supplemental Table S3 - Lineage-associated gene expression validation: Microarray-Q-RT-PCR correlation.
  • Supplemental Table S4A - eSF and eMSC lineage-associated genes expressed in FACS-isolated control eSF and derived early and late cultures (compared to eMSC FACS.control).
  • Supplemental Table S4B - eSF and eMSC lineage-associated genes expressed in FACS-isolated endometriosis eSF and derived early and late cultures (compared to eMSC FACS.endo).
  • Supplemental Table S5A - eMSC and eSF lineage-associated genes expressed in FACS-isolated control eMSC and derived early and late cultures (compared to eSF FACS.control).
  • Supplemental Table S5B - eMSC and eSF lineage-associated genes expressed in FACS-isolated endometriosis eMSC and derived early and late cultures (compared to eSF FACS Endo).
  • Supplemental Table S6 - Pathway analysis: Altered eMSC and eSF cellular functions in endometriosis vs. control in vivo and in vitro.
  • Supplemental Table S7 - Common differentially expressed genes in control eSF FACS and eSF- and eMSC-derived late cultures vs eMSC FACS control.