Glycine N-methyltransferase deletion in mice diverts carbon flux from gluconeogenesis to pathways that utilize excess methionine cycle intermediates

Supporting Information

• Table S1 - Hepatic Phosphatidylethanolamine (PE), Phosphatidylcholine (PC), Diacylglycerol, Ceramide, and Sphingolipid Metabolites.
• Figure S1 - Schematic representation of experimental procedures and timeline.