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. Author manuscript; available in PMC: 2016 Apr 8.
Published in final edited form as: J Physiol. 2016 Feb 4;594(14):3981–3992. doi: 10.1113/JP271588

Figure 3. The anti-fibrillatory effect of carbamylcholine is dependent upon stimulation of the nicotinic and muscarinic receptors.

Figure 3

A. Carbamylcholine (CCh, 200nmol/L) results in a significant increase in ventricular fibrillation threshold (VFT), which is reversed upon washout of the drug (*p<0.05, n=9) B. This effect is abolished by blockade of the muscarinic receptor with atropine (0.1μmol/L, n=6) and C. nicotinic receptor with mecamylamine (Mec, 10μmol/L, n=6).