Table 1.

The ten patient cases where panel predicted visual field were consistent with SVOP results. Patients with abnormal visual fields are listed before those with normal fields. Details included are (1) Patient demographics (diagnosis and procedures prior to SVOP test and age at SVOP test), (2) Ophthalmology assessment outcomes (visual acuity and confrontation and Goldmann perimetry if attempted), (3) Neuroimaging outcomes (a scan image and subsequent panel predicted visual field), and (4) The SVOP test outcomes (SVOP plot and visual field description).

Abbreviations: VA (visual acuity), NPL (no perception of light), PL (perception of light), NF1 (Neurofibromatosis type 1), SVOP (Saccadic Vector Optokinetic Perimetry).

	Patient demographics		Ophthalmology assessment outcomes			Neuro-imaging outcomes		SVOP outcomes
Case	Diagnosis and procedures prior to SVOP test	Age at SVOP test (years)	VA Right	VA Left	Confrontation and/or Goldmann visual field	Imaging description	Panel predicted visual field on binocular testing	SVOP description
1.	1. Left optic nerve/hypothalamic pilocytic astrocytoma 2. Biopsy at diagnosis 3. Chemotherapy completed October 2006	5.5	6/9	NPL	Confrontation (April 2008) Complete temporal hemianopia of right eye	(July 2008) T1 post gadolinium axial image showed suprasellar hypothalamic enhancing lesion adjacent to left chiasm	Right hemianopia, may have some residual right sided function	Binocular (May 2008) Right superior quadrantanopia
Table 1. The ten patient cases where panel predicted visual field were consistent with SVOP results (continued).
2.	1.Right optic nerve/hypothalamic pilocytic astrocytoma 2.Right frontal craniotomy with subtotal removal January 2008	2.9	PL	6/9	Confrontation Left eye complete temporal defect. Right eye “impossible” to test (October 2008)	(August 2008) T1 post gadolinium axial image showed residual postoperative suprasellar cystic lesion with enhancing soft tissue abutting right chiasm and right internal carotid artery	Left hemianopia, could have subtle right visual field loss in addition	Binocular (October 2008) Left temporal hemianopia with missed points right inferior quadrant
4.	1. NF1 2. Spectacles for accommodative esotropia 3. Optic chiasm glioma	3.8	6/9	6/9	No information available	(July 2012) Coronal FLAIR image showed asymmetric thickening of optic nerves and chiasm with extension into left thalamus	Normal field, could have patchy loss	Binocular (August 2012) Inferior scattered loss
5.	1. Right parieto-occipital high grade glioma 2. Surgical resection September 2011 3. Focal cranial radiotherapy completed November 2011	5.1	6/6	6/6	Confrontation fields full. (October 2011) Goldmann unable to perform (January 2012)	(January 2012) T2 axial image showed Surgical resection cavity in right parieto-occipital lobe	Left hemianopia, could have superior sparing	Binocular (December 2011) Left inferior quadrantanopia
Table 1. The ten patient cases where panel predicted visual field were consistent with SVOP results (continued).
7.	1. Hypothalamic ependymoma 2. Fronto-temporal craniotomy and debulking of left suprasellar mass July 2009	3.2	6/6	NPL	Confrontation Difficult to test visual function (February 2010)	(September 2009) T1 post gadolinium axial image showed prominent right optic nerve with residual tumour in suprasellar cistern	Right hemianopia, could have some left sided loss	Binocular (November 2009) Right hemianopia and random scattered left hemifield missed points.
14.	1. Left fronto-temporal anaplastic ependymoma 2.Craniotomy and excision of tumour December 2009; subsequent repeat craniotomy and excision of recurrence March 2010 3. Cranial radiotherapy	6.3	6/5	6/9	Confrontation Examination normal (May 2011)	(April 2011) T1 post gadolinium axial image showed evidence of previous surgery and radiotherapy in left temporal lobe	Right hemianopia, may have inferior sparing	Left eye (March 2011) Scattered superior and nasal loss on left monocular visual field test. Right monocular and binocular visual field both normal.
15.	1. Hypothalamic pilocytic astrocytoma 2. Biopsy and right ventriculoperitoneal (VP) shunt April 2010; Left VP shunt August 2010 3. Focal radiotherapy November 2010	15.0	3/36	6/5	Goldmann showed incomplete left homonymous hemianopia. Some residual vision to left of vertical midline (June 2011)	(January 2011) T1 post gadolinium axial image showed hypothalamic tumour with central necrosis and peripheral enhancement post-radiotherapy	Left hemianopia	Left eye (February 2011) Left hemianopia
Table 1. The ten patient cases where panel predicted visual field were consistent with SVOP results (continued).
16.	1. Left temporal pilocytic astrocytoma 2. Left frontotemporal craniotomy and debulking September 2009	4.4	6/5	6/6	Confrontation showed signs of right homonymous hemianopia (October 2009)	(September 2009) T2 axial image showed left temporal resection cavity with medial extension of residual tumour in left thalamus and compression of chiasm	Right hemianopia	Binocular (October 2009) Right hemianopia with missed points on left side
3.	1. NF1 2. Right optic tract thickening - possibly small glioma, T2 hyperintensity Left internal capsule 3. Poor motor co-ordination and dyspraxia	10.7	6/6	6/6	Goldmann Within normal limits (April 2012)	(September 2011) Coronal FLAIR image showed T2 hyperintensities in the globus pallidus bilaterally, in keeping with NF1	Normal field	Binocular (February 2012) Normal field
6.	1. Posterior fossa ependymoma - mainly L cerebellar pontine angle 2. Posterior fossa craniotomy and complete excision November 2009 3. Proton beam radiotherapy completed March 2010	5.2	6/6	6/6	Confrontation fields full (June 2010)	(January 2011) T1 post gadolinium axial image showed left posterior fossa surgical resection cavity	Normal field	Binocular (January 2011) Normal field