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. Author manuscript; available in PMC: 2018 Oct 5.
Published in final edited form as: Biochim Biophys Acta Gene Regul Mech. 2018 Oct 10;1861(10):952–961. doi: 10.1016/j.bbagrm.2018.08.006

Fig. 2. Effect of CckA(L228P) mutation on CtrA.

Fig. 2

(A) In vivo phosphorylation experiment denoting CtrA~P/CtrA levels in WT, WT cckA(L228P), ∆nstA and ∆nstA cckA(L228P) mutants. (B) Data of qChIP analysis showing the CtrA occupancy at the promoter region of pilA (PpilA) in WT, WT cckA(L228P), ∆nstA and ∆nstA cckA(L228P) cells. The relative β-galactosidase activity (in percentage) of (C) the PpilA-lacZ and (D) the PtacA-lacZ reporters in WT, WT cckA(L228P), ∆nstA and ∆nstA cckA(L228P) cells. The values ± SE, represented in A, B, C and D are the average of at least three independent experiments.