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. Author manuscript; available in PMC: 2019 May 16.
Published in final edited form as: Cardiovasc Drugs Ther. 2019 Jan 8;33(2):221–230. doi: 10.1007/s10557-018-6845-3

Fig. 4.

Fig. 4

The accumulation of cMyC vs. cTnT after myocardial injury caused by intracoronary ethanol. Venous blood was collected frequently over the first 2 h, and up to 24 h, after therapeutic alcohol septal ablation for hypertrophic cardiomyopathy (TASH) using ethanol infused selectively into a septal perforating branch coronary artery. Summary data of absolute quantification of cMyC (open symbols) vs. cTnT (closed symbols) over time following TASH (n = 20). Inset figure is a zoom of the first 240 min. Over this time interval, cMyC accumulates in the serum approximately six times faster than cTnT (slope 25.8 ± 1.9 vs. 4.0 ± 0.4 ng/L/min, p < 0.0001). Figure reproduced from Baker et al. [22]