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. Author manuscript; available in PMC: 2020 Mar 2.
Published in final edited form as: FASEB Bioadv. 2019 Aug 23;1(10):609–623. doi: 10.1096/fba.2019-00048

Fig. 2. Acute exposure to inhaled e-cigarette aerosols containing nicotine caused pro-inflammatory mediator release in BALF.

Fig. 2.

Mice were exposed to air, PG, and PG with nicotine (PG+Nicotine) for 3 days (2 hrs/day). Mice were sacrificed 2 hrs post-last exposure on day 3. Pro-inflammatory mediators in the BALF were measured by Bio-Plex Pro-mouse cytokine 23-plex assay kit (Bio-Rad). Data are shown as mean ± SEM (n=6/group; equal number of male and female mice were used; *P < 0.05, **P < 0.01, ***P < 0.001, significant compared with air control; #P < 0.05, # #P < 0.01, significant compared with PG exposed group.)