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. Author manuscript; available in PMC: 2020 Mar 2.
Published in final edited form as: FASEB Bioadv. 2019 Aug 23;1(10):609–623. doi: 10.1096/fba.2019-00048

Fig. 5. Acute exposure to inhaled e-cig aerosols containing PG with or without nicotine leads to dysregulated lipogenic/myogenic markers in mouse lungs.

Fig. 5.

The protein abundance of lipogenic and myogenic markers were measured in lung homogenates by western blotting. β-actin was used as an endogenous control. Representative blots for lymphoid enhancer binding factor 1 (LEF-1), peroxisome proliferator-activated receptor γ (PPARγ), β-Catenin, and E-cadherin levels in air, PG and PG with nicotine (PG+nicotine) exposed (A) male and (B) female mice are shown. (C) The band intensity was measured by densitometry and data are shown as fold change relative to air group control; β-actin was used as a loading control. Data are shown as mean ± SEM (n=6/group; equal number of male and female mice were used; n=3 for male and female groups respectively; *P < 0.05, **P < 0.01, ***P < 0.001, significant compared with air exposed control.)