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. Author manuscript; available in PMC: 2021 Dec 17.
Published in final edited form as: Neurodegener Dis. 2021 Jul 28;21(1-2):1–23. doi: 10.1159/000518581

Table 2.

Rate estimations of long-term symptoms, loosely organized by frequency, from clinical patients recovered from acute features of COVID-19. A similar reference population is compared with to disentangle the effects of the COVID-19 pandemic from SARS-CoV-2 infection. Only symptoms that could have a primary or secondary effect on cognition are included.

Clinical COVID-19 survivor Comparison population
≥ 1 Symptom 50% [122], 76% [151] -
Chest CT abnormalities 71% [152], 53% [151] -
Fatigue 53%** [123], 28-53% [122,124], 63% [151] 9-22% [122,153]
Myocardial inflammation 30-60% [154,155] -
Psychomotor coordination impairment 57% [127] -
Executive function impairment 50% [127] -
PTSD 28%** [126] 3-17% [156,157]
Loss of concentration 26%** [125], 33% [127] -
Verbal fluency loss 32% [127] -
Insomnia/sleep disturbance 8-40%** [125,126], 18% [122], 26% [151] 5-32% [122,156]
Depression 21-31%** [125,126], 4.3% [122], 23% [151] 1-28% [122,156,157]
Memory loss 18%** [125], 24% [127] -
Pulmonary function abnormalities 21-58% [122,158], 22-56% [151] 5% [122]
Resting heart rate increase 11% [122] 0% [122]
Hematuria (kidney) 57%* [159] -
Proteinuria (kidney) 31%* [159] -
Liver ALT, AST/ALT, GGT & ALP Levels   *[160] -
*

= Measurements at Hospital Discharge.

**

= Measurements at 1 month.

= Measurements at 3 months.

= Measurement at 6 months.