Skip to main content
. Author manuscript; available in PMC: 2022 Jun 4.
Published in final edited form as: J Comp Neurol. 2022 Jan;530(1):6–503. doi: 10.1002/cne.25243

Fig. 15.

Fig. 15.

CALB2 expression in the thalamus at PCW 21. (a-g) Sequential sections showing CALB2 expression in the anterior ventral (AV) and anteromedial (AM) thalamic nuclei, paraventricular nucleus (PaV), midline nuclear complex (MiN), ventral anterior nucleus (VA), ventral lateral nucleus (VL), centromedian nucleus (CeM), reuniens nucleus (Re), and paracentral nucleus (PC) (a-d). Clear CALB2 expression is also seen in the mediodorsal nucleus (MD), ventromedial nucleus (VM), lateral dorsal nucleus (LD), central lateral nucleus (CL), habenular nucleus (HN), periventricular area (PeVA), subparafascicular nucleus (SPf), lateral posterior nucleus (LP), pulvinar (Pul), dorsal lateral geniculate nucleus (DLG) and limitans/suprageniculate nuclei (LSG) (c-g). In contrast, faint CALB2 expression is observed in ventral posterior lateral (VPL), ventral posterior medial (VPM), parafascicular nucleus (Pf), central medial nucleus (CM), pregeniculate nucleus (PG) and zona incerta (ZI) (e-f). Note the CALB2 expression in the patches within Pf/CM (f). Note also the CALB2 expression in the subthalamic nucleus (STH; c-e) and inter-pallidal transient cell zone (IPTC; a). Strong CALB2 expression is also found in a region that appears to be equivalent to mouse posterior intralaminar nucleus (PIL). Scale bar: 800 μm in (a) for all panels.