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. Author manuscript; available in PMC: 2010 May 25.
Published in final edited form as: J Neurosci. 2009 Nov 25;29(47):14903–14911. doi: 10.1523/JNEUROSCI.3436-09.2009

Figure 3. Phosphorylation of Ser293 on Cx36 does not alter trafficking or distribution of the protein.

Figure 3

A, Conditions that altered Cx36 phosphorylation at Ser293 (D1R activation, SKF38393, 100 µM; D1R antagonism, SCH23390, 100 µM) had no effect on the size of Cx36 plaques in AII amacrine cells. Inhibition of PKA (Rp-8-CPT-cAMPS, 20 µM) also had no effect on Cx36 plaque size. B, Conditions that altered Cx36 phosphorylation at Ser293 (D1R activation, SKF38393, 100 µM; D1R antagonism, SCH23390, 100 µM) had no effect on the overall distribution of Cx36 plaques in AII amacrine cells. Inhibition of PKA (Rp-8-CPT-cAMPS, 20 µM) also had no effect on Cx36 plaque distribution. Error bars are s.e.m, n = 5. No significant changes were found.