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. Author manuscript; available in PMC: 2013 Jun 28.
Published in final edited form as: J Neurosci. 2000 Feb 1;20(3):929–936. doi: 10.1523/JNEUROSCI.20-03-00929.2000

Figure 2.

Figure 2

Effect of SRIF on delayed outward K+ current (IK) in rods. A, SRIF increased voltage-activated K+ current. IK was evoked by holding cells at −70 mV and applying depolarizing steps from −60 to +40 mV in 20 mV increments in control external solution (left) and after a 4 min exposure to 0.5 μM SRIF (striped horizontal bar; right). B, I–V relationship of IK obtained from the experiment described in A is shown. Inset, Somatostatin increased IK amplitude without substantially changing the current–voltage relation. □, Control; ○, somatostatin. The y-axis of the inset is normalized current. C, CTX reduced outward currents (left) but did not prevent a somatostatin-induced increase in IK. D, I–V relationship of outward current in the presence of CTX (□), or CTX + somatostatin (△); n = 3.