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. Author manuscript; available in PMC: 2010 Jul 27.
Published in final edited form as: Pharmaceuticals (Basel). 2010 Apr 1;3(4):1093–1107. doi: 10.3390/ph3041093

Figure 1.

Figure 1

Perfusion of intact ventricular cardiomyocytes with a high concentration (30 μM) of IpTxa causes immediate cessation of [Ca2+]i transients. (A) line-scan image (top) and associated fluorescence plot (bottom) of a field-stimulated mouse ventricular cardiomyocyte loaded with the Ca2+ indicator Fluo-4 and perfused constantly with normal Tyrodes solution in the absence of IpTxa (control). (B) The same cell ~1 min after perfusion with the same Tyrodes solution, but containing 30 μM IpTxa. The protocol was repeated four times and the result below is representative of three experiments.