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. Author manuscript; available in PMC: 2013 Apr 3.
Published in final edited form as: J Neurosci. 2012 Oct 3;32(40):13718–13728. doi: 10.1523/JNEUROSCI.5750-11.2012

Fig. 7. Chronic dopamine depletion increased the number bassoon-immunoreactive structures associated with large vGAT-immunoreactive axon terminals.

Fig. 7

Representative through-focus confocal micrographs of bassoon (green) and vGAT (red) co-immunoreactive (yellow) axon terminals in the STN of control (A–C) and 6-OHDA-lesioned (D–E) animals. 3 examples (A–C and D–F) per condition are illustrated. The number of bassoon-immunoreactive structures per vGAT-immunoreactive axon terminal (arrows) was significantly increased by chronic dopamine depletion compared to control both in the representative examples and across the sample population (G). The scale bar in A applies to each micrograph. *, p < 0.05.