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. Author manuscript; available in PMC: 2014 Apr 9.
Published in final edited form as: J Innate Immun. 2013 Apr 9;5(6):543–554. doi: 10.1159/000347060

Figure 6.

Figure 6

Recipient mice developed a stronger allergic airway inflammation when sensitized by OVA/UFP-exposed Nrf2-/- DC via adoptive transfer. Adoptive transfer and secondary OVA aerosol challenge were performed as described in the Materials and Methods. A. BAL cellular differentiation showing that sensitization by OVA/UFP-treated Nrf2-/- DC led to eosinophilic inflammation in the lungs, *p < 0.01 compared with respective saline control, ap < 0.01 compared with respective OVA group. B. Increased OVA-IgG1 in the blood of recipient mice sensitized by OVA/UFP-exposed DC, *p < 0.02 compared with all other groups. C. Elevated IL-13 level in the BAL fluid, *p < 0.01 compared with respective saline control, ap < 0.05 compared with respective OVA group.