. Author manuscript; available in PMC: 2014 Nov 4.

Published in final edited form as: IEEE J Transl Eng Health Med. 2013 Oct 16;1:1500210. doi: 10.1109/JTEHM.2013.2285916

Table I.

The effects of metabolic inhibitors and uncoupler on the lung tissue surface FAD and NADH fluorescence signals of normoxic and hyperoxic rats.

Inhibitor/Uncoupler	FAD %		NADH %
Inhibitor/Uncoupler	Normoxic	Hyperoxic	Normoxic	Hyperoxic
Rotenone	0.2 ± 0.2	−0.3 ± 0.3	20.2 ± 2.3	7.5 ± 1.0^*
KCN	−6.8 ± 1.6	−5.2 ± 0.5	21.7 ± 2.5	9.2 ± 1.2^*
PCP	1.7 ± 0.8	−2.3 ± 1.4^*	−19.7 ± 2.1	−20.8 ± 1.2
PCP + KCN	−9.3 ± 1.0	−4.2 ± 0.7^*	25.7 ± 2.6	9.0 ± 1.0^*
Rotenone + KCN	−3.3 ± 0.8	−3.8 ± 0.6	4.6 ± 1.0	2.8 ± 1.2

Values are mean ± SE. For the (PCP + KCN) and (rotenone + KCN) treatments, the percentage change due to additional treatment with KCN is calculated relative to the new baseline in the presence of PCP or rotenone, respectively. For normoxic lungs, n = 5, 7, 6, 6, and 5 for rotenone, potassium cyanide (KCN), pentachlorophenol (PCP), PCP + KCN, and rotenone + KCN, respectively. For hyperoxic lungs, n = 4, 5, 6, 5, and 4 for rotenone, KCN, PCP, PCP + KCN, and rotenone + KCN, respectively.

indicates value significantly different from the corresponding normoxic value (t-test, P < 0.05).