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. Author manuscript; available in PMC: 2009 Nov 12.
Published in final edited form as: J Neurosci. 2008 Nov 12;28(46):11806–11813. doi: 10.1523/JNEUROSCI.3796-08.2008

Figure 1.

Figure 1

Neuromodulation of the crustacean gastric mill CPG network states. Computational modeling shows that neuromodulation (by the peptide pyrokinin: PK) of the gastric mill rhythm elicited by the projection neuron MCN1 can result in little qualitative change in network output (top traces). Yet, in contrast to the unmodulated or weakly modulated state (A), the generation of network oscillations in the strongly modulated state (B) does not depend on presynaptic inhibition of the axon terminals of the projection neuron MCN1 (bottom traces). Models modified from Kintos et al (2008).