Skip to main content
. Author manuscript; available in PMC: 2017 Oct 20.
Published in final edited form as: JACC Basic Transl Sci. 2017 Apr 24;2(2):160–180. doi: 10.1016/j.jacbts.2016.12.002

Figure 10. Induced PP2Ce expression promotes cardiomyocyte death in response to ischemia/reperfusion injury.

Figure 10

A. Representative TTC staining cross-section images from one PP2Ce transgenic (TG) and one control (control) heart following 15 minutes of ischemia and 120 minutes of reperfusion (I/R). B. Infarct sizes of control (n=5) and PP2Ce transgenic (TG, n=6) hearts as measured from A. **: p<0.01 between control and PP2Ce TG groups. C. Apoptotic index measured by TUNEL staining in control (n=4) and PP2Ce TG (n=4) hearts. **: p<0.01 between corresponding control and PP2Ce TG groups. D. Representative immunoblot for level of activated caspase9 in control (n=10) and PP2Ce transgenic (TG, n=8) hearts following the same I/R protocol. E. Quantification of level of cleaved caspase9 signal from immunoblots. *: p<0.05 between control and PP2Ce TG groups.