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. Author manuscript; available in PMC: 2018 Oct 17.
Published in final edited form as: Molecules. 2018 Sep 13;23(9):E2338. doi: 10.3390/molecules23092338

Figure 6.

Figure 6.

Kaempferol increased GCK activity and glycogen contents in STZ-induced diabetic mice. Diabetic mice were treated with kaempferol for 12 weeks. (a, e) Hepatic GCK, and (b, e) GCKRP protein levels were measured by Western blot and normalized to total protein contents. Hexokinase activity (c) and glycogen content (d) in the liver were measured as described in the Method section. (f) Glucose oxidation was evaluated in fresh mouse liver homogenates using 14C-labeled glucose as described in the Method section. Data are shown as Mean ± SEM (n=8–9). *, P<0.05 vs. non-diabetic mice; # vs. STZ-induced diabetic mice. Control: nondiabetic mice; STZ: STZ-induced diabetic mice: STZ+Kaemp: STZ-induced diabetic mice treated with kaempferol (50mg/kg BW).