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. Author manuscript; available in PMC: 2023 Jul 12.
Published in final edited form as: Immunity. 2022 May 27;55(7):1200–1215.e6. doi: 10.1016/j.immuni.2022.05.003

Figure 6. IL-21-dependent CX3CR1+ exhausted CD8+ T cells in LCMV-c13-infected mice are maintained in the absence of Bcl6 in CD4+ T cells. (See also Figure S6).

Figure 6.

(A, B) Expression of PD-1, TCF-1 and CX3CR1 in splenic LCMV-gp33-specific CD8+ T cells from LCMV-c13-infected MuMT mice on 21 dpi. Representative plots (A) and pooled data from 2 experiments with n = 2–3 / genotype /experiment shown with mean±SD. Unpaired t-test.

(C, D) Expression of PD-1, TCF-1 and CX3CR1 in splenic LCMV-gp33-specific CD8+ T cells from LCMV-c13-infected Cd40lg-cre Bcl6F/F mice on 21 dpi. Representative plots (C) and pooled data from 2 experiments with n = 2–3 / genotype /experiment shown with mean±SD. Unpaired t-test.