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. 2022 Aug 16;179(17):4458. doi: 10.1111/bph.15929

Erratum

PMCID: PMC10117620  PMID: 35971962

This article corrects the following: https://doi.org/10.1111/bph.15751

In the paper of Lacalle‐Aurioles, M, Trigiani, LJ, Bourourou, M, et al. Alzheimer's disease and cerebrovascular pathology alter inward rectifier potassium (KIR2.1) channels in endothelium of mouse cerebral arteries. Br J Pharmacol. 2022; 179: 2259–2274, https://doi.org/10.1111/bph.15751, an error was introduced in Figure 7 during the production of the printed version of the paper, which altered panel (b) on the Figure.

The correct legend (baseline and Ba2+) for panel (b) on Figure 7 and legend are provided below.

FIGURE 7.

FIGURE 7

Inflammation alters KIR channel‐mediated haemodynamic responses and electrophysiology to whisker stimulation. (a) No effect of Ba2+ (10 μM) on haemodynamic responses to whisker stimulation in WT mice (n = 5). (b) Significantly reduced cerebral blood flow (CBF) and oxyhaemoglobin responses (peak HbO) in WT but not in TGF mice to whisker stimulation after superfusion with Ba2+ (100 μM). (c) Deoxyhaemoglobin (HbR) was reduced in TGF mice after Ba2+ superfusion. Conserved neuronal activity after superfusion with Ba2+ (100 μM) in WT mice but not in TGF mice (showing reduced θ, α, and γ power). *P < 0.05 represent differences between baseline and treatment in Student's t test. Error bars represent SEM. n = 9 for WT; n = 10 for TGF. HbT, total haemoglobin

We apologize for this error.


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