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The Texas Heart Institute Journal logoLink to The Texas Heart Institute Journal
. 2001;28(4):322–323.

Spontaneous Echo Contrast in Left Atrial Appendage during Sinus Rhythm

Mumtaz A Siddiqui 1, Mark J Holmberg 1, Ijaz A Khan 1
Editor: Raymond F Stainback2
PMCID: PMC101214  PMID: 11777163

Spontaneous echo contrast is an echogenic swirling pattern of blood flow, distinct from white noise artifacts, caused by an increased ultrasonic back-scatter from aggregation of the cellular components of blood in the conditions of blood stasis or low-velocity blood flow. 1,2 Although this phenomenon frequently develops in a dilated and dysfunctional left atrium and left atrial appendage during atrial fibrillation, it can develop in any cardiac chamber during any rhythm if the blood flow velocity is markedly low. 3,4 The presence of spontaneous echo contrast in the left atrium and left atrial appendage has been associated with a higher risk of thromboembolism and cerebrovascular accident. 5,6

A transesophageal echocardiogram was performed to evaluate a mitral valve prosthesis in a 51-year-old man. The patient was taking warfarin, and the international normalized ratio (INR) was 3.4 when we performed transesophageal echocardiography. The patient was in sinus rhythm. The transesophageal echocardiographic examination revealed a dilated left atrium and left atrial appendage. The left atrial appendage was multilobulated and contained homogenous finger-like pectinate muscles and spontaneous echo contrast (Figs. 1 and 2). No thrombi were seen in the left atrium or in the left atrial appendage. The contractile function of the left atrial appendage was markedly decreased, with a peak left atrial appendage systolic velocity of 15 cm/sec (normal, ≥40 cm/sec) on Doppler evaluation.

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Fig. 1 Mid-esophageal echocardiographic view of the left atrium and left atrial appendage in a 123° plane shows spontaneous echo contrast.

Real-time motion image is available at www.texasheartinstitute.org/siddiqui284.html.

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Fig. 2 Diagram illustrates various components of Figure 1.

1 = left atrium; 2 = left atrial appendage; 3 = pectinate muscles; 4 = lobules of left atrial appendage; 5 = spontaneous echo contrast; 6 = pulmonary trunk

Supplementary Material

Video for Fig. 1
Download video file (529.5KB, mpg)

Footnotes

Web site: This article has also been published on the THI Web site at www.texasheartinstitute.org/siddiqui284.html

Address for reprints: Ijaz A. Khan, MD, Creighton University Cardiac Center, 3006 Webster Street, Omaha, NE 68131-2044

References

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  • 2.Sigel B, Coelho JC, Spigos DG, Flanigan DP, Schuler JJ, Kasprisin DO, et al. Ultrasonography of blood during stasis and coagulation. Invest Radiol 1981;16:71–6. [DOI] [PubMed]
  • 3.Beppu S, Nimura Y, Sakakibara H, Nagata S, Park YD, Izumi S. Smoke-like echo in the left atrial cavity in mitral valve disease: its features and significance. J Am Coll Cardiol 1985;6:744–9. [DOI] [PubMed]
  • 4.Black IW, Hopkins AP, Lee LC, Walsh WF. Left atrial spontaneous echo contrast: a clinical and echocardiographic analysis. J Am Coll Cardiol 1991;18:398–404. [DOI] [PubMed]
  • 5.Daniel WG, Nellessen U, Schroder E, Nonnast-Daniel B, Bednarski P, Nikutta P, Lichtlen PR. Left atrial spontaneous echo contrast in mitral valve disease: an indicator for an increased thromboembolic risk. J Am Coll Cardiol 1988;11:1204–11. [DOI] [PubMed]
  • 6.Fatkin D, Kelly RP, Feneley MP. Relations between left atrial appendage blood flow velocity, spontaneous echocardiographic contrast and thromboembolic risk in vivo. J Am Coll Cardiol 1994;23:961–9. [DOI] [PubMed]

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Supplementary Materials

Video for Fig. 1
Download video file (529.5KB, mpg)

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