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. 2023 Feb 24;10(2):630–631. doi: 10.1016/j.gendis.2023.02.005

Corrigendum to “Establishment and functional characterization of the reversibly immortalized mouse glomerular podocytes (imPODs)” [Genes & Diseases 5 (2018) 137–149]

Xinyi Yu a,b, Liqun Chen a,b, Ke Wu b,c, Shujuan Yan b,c, Ruyi Zhang b,c, Chen Zhao a,b, Zongyue Zeng b,c, Yi Shu b,c, Shifeng Huang a,b, Jiayan Lei a,b, Xiaojuan Ji a,b, Chengfu Yuan b,d, Linghuan Zhang b,c, Yixiao Feng a,b, Wei Liu a,b, Bo Huang b,c,e, Bo Zhang b,f, Wenping Luo b,c, Xi Wang b,c, Bo Liu a,b, Rex C Haydon b, Hue H Luu b, Tong-Chuan He b, Hua Gan a,
PMCID: PMC10201652  PMID: 37223498

The authors regret having an image assembly error in Figure 3A, in which the image for “imPOD Synaptopodin DAPI stain” group was erroneously duplicated with the image from the “tsPOD-33 °C Synaptopodin DAPI stain” group.

We confirm the error is restricted to the image assembly, and the underlying data and conclusions are correct and unchanged.

The authors would like to apologize for any inconvenience caused.

Image 1

Figure 3 The imPOD cells express podocyte markers. The imPOD and parental tsPC (at 33 °C + 100U/ml γ-interferon) cells were seeded at subconfluence and stained with antibodies against the podocyte specific marker (WT-1) and Synaptopodin (A), as well as the slit diaphragm complex related markers (Nephrin, Tubulin and Vinculin) (B). Stains without primary antibodies were used as negative controls. Cell nuclei were counter-stained with DAPI. Representative images are shown.

Footnotes

Peer review under responsibility of Chongqing Medical University.


Articles from Genes & Diseases are provided here courtesy of Chongqing Medical University

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