Correction: Tobacco smoke induced hepatic cancer stem cell-like properties through IL-33/p38 pathway

Correction: J Exp Clin Cancer Res 38, 39 (2019)

https://doi.org/10.1186/s13046-019-1052-z

Following publication of the original article [1], an overlapping of images was identified Fig. 1e and Fig. 5b.

The images of Oct4 in TS and TS + DMSO groups were replaced and the correct Fig. 5 is given as below:

Fig. 5.

p38 suppression reversed long term TS exposure-triggered CSC-like properties. Mice exposed to TS were treated with or without p38 MAPK inhibitor (SB 203580) for 12 weeks, and representative micrographs of liver tissue were stained with H&E (a). b Immunohistochemical staining for EpCAM, CD133 and Nanog, Oct4 in liver tissues. c-f Fold changes of EpCAM (c), CD133 (d), Nanog (e) and Oct4 (f)—positive area in TS group compared with FA group. g Western blotting of EpCAM and CD133 in liver tissues. β-actin was served as the loading control. h-i The indicated proteins relative to β-actin were assessed by densitometric analysis; six animal samples per group were used for the densitometric analysis. Data are expressed as mean ± SD. The significance was assessed with one-way ANOVA test. # P < 0.05, ## P < 0.01, compared with FA control; * P < 0.05, ** P < 0.01, compared with TS + DMSO group. FA = filtered air; TS = tobacco smoke

The correction does not affect the overall results or conclusion of the article.