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The authors inadvertently inserted an incorrect graph in Fig. 3C. A corrected version of the figure and figure legend is presented below. The authors apologize for this error and state that the correction does not change the scientific conclusions of the article.
Figure 3.
PG e-cig aerosols induce mucociliary dysfunction in primary HBECs in vitro. A: representative schema illustrating exposure of fully differentiated primary HBECs to e-cig aerosols. Arrows indicate direction of aerosol flow. B: mucus concentrations are significantly increased in HBECs exposed to PG e-cig aerosols after 5 days compared with air-exposed controls. n = 6 from 5 lungs. C: ciliary beat frequency (CBF) of HBECs exposed to PG e-cig aerosols is significantly reduced after 5 days compared with air-exposed controls. n ≥ 36 from 4 lungs. D: 5-day exposure of HBECs to PG e-cig aerosols does not affect cytotoxicity as assessed by lactate dehydrogenase (LDH) release into basolateral media. n = 6 lungs. Data are presented as means ± SE. *P < 0.05, ns = not significant. Data were analyzed by two-tailed t test (B and D) or mixed-effects model (C) after assessing normality by Shapiro–Wilk. HBECs, human bronchial epithelial cells; PG, propylene glycol.