The authors wish to make the following corrections to this paper [1]:
In Section 1: Introduction, paragraph 4, lines 1–2, the author mistakenly wrote: “Dogs are excellent experimental animal models in many types of biomedical research studies, and most diseases in dogs are homologous to humans [30–33]”.
The correct version should be as follows:
“Dogs are the experimental animal model used in many types of biomedical research studies [30], and some diseases in dogs are homologous to humans [31–33]. However, there is also growing evidence that the dog model is often not sufficiently justified and characterized as a relevant model for the human disease being studied [30].”
The reference order will also be updated accordingly.
The authors apologize for any inconvenience caused and state that the scientific conclusions are unaffected. The original publication has also been updated.
Footnotes
Disclaimer/Publisher’s Note: The statements, opinions and data contained in all publications are solely those of the individual author(s) and contributor(s) and not of MDPI and/or the editor(s). MDPI and/or the editor(s) disclaim responsibility for any injury to people or property resulting from any ideas, methods, instructions or products referred to in the content.
Reference
- 1.Mao A., Zhao W., Zhu Y., Kong F., Chen D., Si H., Xu C. Gut Bacterial Community Determines the Therapeutic Effect of Ginsenoside on Canine Inflammatory Bowel Disease by Modulating the Colonic Mucosal Barrier. Microorganisms. 2023;11:2616. doi: 10.3390/microorganisms11112616. [DOI] [PMC free article] [PubMed] [Google Scholar]