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Published in final edited form as: J Popul Econ. 2020 Mar 16;33:1499–1522. doi: 10.1007/s00148-020-00772-8

Ethnic attrition, assimilation, and the measured health outcomes of Mexican Americans

Francisca M Antman 1, Brian Duncan 2, Stephen J Trejo 3
PMCID: PMC10846883  NIHMSID: NIHMS1913457  PMID: 38323118

Abstract

The literature on immigrant assimilation and intergenerational progress has sometimes reached surprising conclusions, such as the puzzle of immigrant advantage which finds that Hispanic immigrants sometimes have better health than US-born Hispanics. While numerous studies have attempted to explain these patterns, almost all studies rely on subjective measures of ethnic self-identification to identify immigrants’ descendants. This can lead to bias due to “ethnic attrition,” which occurs whenever a US-born descendant of a Hispanic immigrant fails to self-identify as Hispanic. In this paper, we exploit information on parents’ and grandparents’ place of birth to show that Mexican ethnic attrition, operating through intermarriage, is sizable and positively selected on health, making subsequent generations of Mexican immigrants appear less healthy than they actually are. Consequently, conventional estimates of health disparities between Mexican Americans and non-Hispanic whites as well as those between Mexican Americans and recent Mexican immigrants have been significantly overstated.

Keywords: Assimilation, Immigrant health advantage, Ethnic attrition, Hispanic health paradox

JEL classification: J15, J12, I14

1. Introduction

There is now a vast literature documenting the health of immigrants and their US-born descendants. One strand of this literature centers on long-term integration and forms a key element in the heated debate over immigration policy, documenting disparities between US-born Hispanics and non-Hispanic whites on measures of health (Hummer et al. 2007; Antecol and Bedard 2006), as well as other measures of human capital (Borjas 1994; Smith 2003). A second strand of the immigrant health literature centers on the existence of an immigrant health advantage, exploring whether 1st-generation Hispanic immigrants are in better health than US-born Hispanics (Singh and Siahpush 2002; Bates et al. 2008; Acevedo-Garcia et al. 2005; Tavernise 2013).1 These studies are closely related to the broader literature on the Hispanic Health Paradox, which attempts to explain why Hispanics appear to be in better health than non-Hispanic whites on some, though not all, measures of health, despite having lower socioeconomic status (Riosmena et al. 2015).

While the literature has explored the roles of selective migration (Palloni and Arias 2004; Riosmena et al. 2013) and regression toward the mean (Giuntella 2017) in explaining the Hispanic Health Paradox more broadly,2 the predominant explanation is the negative acculturation hypothesis (Escarce et al. 2006; National Research Council 2006). According to this view, immigrants arrive in the USA with habits and customs that are more protective of health and gradually assimilate to less-healthy US norms (Antecol and Bedard 2006; Riosmena et al. 2015). Ironically, this means that just as the process of intergenerational assimilation may lead to convergence in health status between US-born Hispanics and non-Hispanic whites, assimilation may also erode any health advantage held by 1st-generation immigrants.

Importantly, the literature exploring these questions relies almost universally on self-reported ethnic identity to identify Hispanic individuals. In contrast, several recent studies have emphasized the importance of distinguishing between self-identification and ancestry in making comparisons across generations and ethnic groups (Alba and Islam 2009; Duncan and Trejo 2011a, 2011b, 2016, 2017; Antman and Duncan 2015). Most significantly, Duncan and Trejo (2011a) document significant rates of Mexican “ethnic attrition” which occurs whenever a US-born descendant of a Mexican immigrant fails to self-identify as Mexican. Moreover, this pattern of ethnic attrition is found to be selective on socioeconomic status (SES): the descendants of Mexican immigrants who do not identify as Mexican are more educated than those who self-identify, resulting in a downward bias in estimates of intergenerational educational progress for Mexican Americans.

This paper explores whether ethnic attrition is also selective on health and how that selection process may bias perceptions of long-term integration vis-à-vis non-Hispanic whites and estimates of any 1st-generation health advantage compared with US-born Hispanics. To focus the analysis, we restrict attention to the experience of Mexicans in particular, as they overwhelmingly form the largest Hispanic origin group in the USA (Motel and Patten 2012). Thus, we explore the extent to which focusing only on groups of self-identified Mexicans gives an incomplete picture of intergenerational health profiles of all Mexican Americans by obscuring the experience of individuals with Mexican ancestry who do not identify as Mexican. Importantly, this problem plagues virtually all health studies of the descendants of immigrants since they rely on subjective measures of ethnic self-identification rather than on more objective measures of lineage. Instead, we use parents’ and grandparents’ place of birth to more objectively identify descendants of Mexican immigrants. Thus, we investigate how any selection into ethnic attrition might bias estimates of the health of US-born Mexicans when the analysis sample consists solely of those who self-identify as Mexican. To explore possible mechanisms, we investigate whether intermarriage is also selective on health and whether intermarriage is linked to the ethnic attrition that we observe.

On the one hand, one might expect a positive correlation between health, intermarriage, and ethnic attrition, since the latter two factors are positively correlated with SES just as health and SES are positively correlated (Adler and Ostrove 2006). Since ethnic attritors approach the SES of non-Hispanic whites more rapidly than those who continue identifying as Mexican, one might also expect health outcomes of ethnic attritors to converge more quickly with those of non-Hispanic whites, at least relative to the path of self-identified Mexicans. Thus, one would expect that focusing only on self-identified Mexicans would make it appear that Mexicans are in worse health than they actually are. This would result in overestimated gaps between US-born Mexicans and non-Hispanic whites, making Mexicans look more poorly assimilated. Assuming a 1st-generation health advantage exists, this would also lead to an overestimated gap between Mexicans immigrants and US-born Mexicans.

On the other hand, Mexican self-identification itself might arguably serve as a measure of acculturation, since individuals with Mexican ancestry who do not identify as Mexican might be more readily identifying with the mainstream non-Hispanic white culture instead. Assuming a 1st-generation health advantage exists at the outset, one might then expect ethnic attritors to make lifestyle choices closer to the mainstream and thus adopt less-healthy lifestyles than individuals who identify as Mexican.3 This type of mechanism would be consistent with the negative acculturation hypothesis. If these less-healthy behaviors are more common among ethnic attritors than non-attritors, we should expect that calculations that exclude individuals based on their lack of Mexican self-identification would make Mexicans appear to be in better health. As a result, we should expect to underestimate the gap between US-born Mexicans and non-Hispanic whites. The same would be true for the gap between Mexicans immigrants and US-born Mexicans, again assuming the health outcome under study implied a 1st-generation advantage.

Since negative acculturation and positive socioeconomic factors are both possible channels contributing to the over- or under-estimates of health premiums assessed over generations and across ethnic groups, we investigate these questions empirically. We find that at higher-order immigrant generations, ethnic attritors display better health outcomes than non-attritors and are closer to those of non-Hispanic whites. We also show that this pattern can be traced to selective intermarriage: marriages between Mexicans and non-Mexicans are more likely to involve spouses in better health and are more likely to produce children that are not identified as Mexican who are also in better health. Moreover, once we control for parental health and SES, the health differential by ethnic attrition disappears, reinforcing the view that the relationship between ethnic attrition and child health is driven by parental characteristics. Together, this collection of results implies that estimates relying on ethnic self-identification bias researchers toward finding that US-born Mexican Americans are in worse health than they actually are. Consequently, conventional estimates of health disparities between Mexican Americans and non-Hispanic whites as well as those between Mexican Americans and recent Mexican immigrants have been significantly overstated.

One important caveat to be noted is that the only measure of health available in our data set, the Current Population Survey (CPS), is self-rated health (SRH) status. This may raise concerns about whether SRH is a good measure for documenting health disparities, which we address directly in Sect. 2.1. In particular, we compare SRH with a wide array of measures used in the health literature, but not available in the CPS, to show that SRH is correlated with health outcomes across immigrant generations. In related work, Antman, Duncan and Trejo (2016) use several arguably more-objective health measures to show that ethnic attritors, defined as children not identified as Mexican American despite having at least one parent identified as Mexican American, are closer to non-Hispanic whites on several measures of health. Those results, using a broader set of health outcomes, are consistent with the results presented here but are limited by the fact that data sets with a richer set of health outcomes only allow for ethnic attrition measures based on parental ethnic self-identification. In contrast, the CPS used in this paper contains information on parental place of birth, thus allowing for a more accurate method of measuring ethnic attrition, and allowing us to precisely identify 3rd-generation immigrants. Thus, this paper addresses the limitations of the earlier work and, taken together, both studies provide a more complete picture of the impact of ethnic attrition on measured health disparities.

Another important caveat to note is that we can only objectively trace immigrant generations as far back as data allow us to link individuals with objective measures of ancestry such as parents’ place of birth and grandparents’ place of birth, which we can obtain for children that still reside with their parents. As we show that ethnic attrition mainly affects immigrants at the 3rd and higher generations, this means that in most of the paper, we analyze health disparities between immigrant children and native children and then investigate the role of ethnic attrition in explaining that difference. This marks another area of contribution for this paper, as most studies of immigrant health disparities focus exclusively on adults. However, our focus on children may also explain why we find neither clear evidence of an immigrant health advantage nor a distinct disadvantage among Mexican-identified children after controlling for socioeconomic status, relative to non-Hispanic white children in our sample. Nevertheless, since our goal is to understand the implications of ethnic attrition for measured health disparities, we are still able to relate these findings back to a sample of adult immigrants and thus contribute to the broader immigrant health literature. To this end, our analysis suggests that both the 1st-generation health advantage and the health disadvantage of later-generation Mexican adults (relative to non-Hispanic whites) are substantially overstated.

Our paper contributes to an emerging literature within economics on ethnic identity and how it relates to immigrant assimilation (Zimmermann 2007; Bisin and Verdier 2011; Constant and Zimmermann 2013). This literature has developed measures of ethnic identity (Constant and Zimmermann 2008; Constant et al. 2009a), investigated the determinants of identity formation and persistence (Bisin and Verdier 2000; Manning and Roy 2010; Masella 2013; Galli and Russo 2019), and analyzed the consequences of ethnic identity for the socioeconomic integration of immigrants and their descendants (Pendakur and Pendakur 2005; Constant et al. 2009b; Constant and Zimmermann 2009; Bisin et al. 2011). This work recognizes the complexity and endogeneity of identity and suggests mechanisms through which ethnic identification could be associated with both observed and unobserved characteristics of individuals. Building on these insights, our paper highlights the complications that intergenerational shifts in ethnic identify can create for measuring the socioeconomic progress of later-generation descendants of immigrants. Our empirical analysis illustrates how such complications play out when attempting to track the health of Mexican Americans across immigrant generations.

The remainder of this paper proceeds as follows. Section 2 describes the data used in the analysis and presents conventional estimates of health disparities across immigrant generations and ethnic groups as they might be displayed in the literature relying on measures of self-identification. Section 3 presents our empirical analyses, which describe how health outcomes for Mexican Americans vary by the nativity and ethnic origins of their ancestors, whether they come from families with mixed ethnic backgrounds, and whether they choose to subjectively identify as Mexican. A discussion of the bias introduced by ethnic attrition follows. Section 4 concludes.

2. Data

The data we use come from the 1996–2017 Current Population Survey (CPS) March Supplement. While the CPS is largely regarded as a labor force survey, covering standard questions about labor force participation and employment outcomes, it also collects important demographic information about respondents and household members that facilitate the analyses below. As mentioned above, we focus our analysis on Mexicans, the largest group of country-specific immigrants to the USA (Motel and Patten 2012).4

2.1. Measuring health status

With regard to health outcomes, the March Supplement collects information on self-rated health (SRH), a measure that is widely used in the literature on immigrant health outcomes (Acevedo-Garcia et al. 2010; Antecol and Bedard 2006; Riosmena et al. 2013). Specifically, respondents are asked to rank the subject’s health on a 5-point scale indicating excellent, very good, good, fair, or poor health status. We follow the literature in assigning a poor health indicator equal to one if the respondent indicates poor or fair health and equal to 0 if the respondent reports that health is good, very good, or excellent.5 Of course, there are understandable concerns about the self-rated health outcome, most notably that it reflects the respondent’s subjective view of health, and thus may differ from objective health status. Nevertheless, it remains widely used throughout the health literature and has been shown to track results using more objective health outcomes such as obesity/body mass index (Antecol and Bedard 2006), predict future disability (Idler and Kasl 1995), and serve as an even better indicator of subsequent mortality than more complex health indicators (Kuhn et al. 2006). Its robust relationship with mortality in particular, documented in a wide variety of settings and replicated in numerous studies, suggests that SRH provides an important dimension of health not captured by other measures (Idler and Benyamini 1997).

At the same time, important questions surround the use of SRH in the current context. First, for reasons discussed below, our analyses focus on children ages 17 and younger. Given their youth, children are naturally less likely to suffer from functional decline and mortality observed in the older populations that have comprised the primary focus in most studies on the link between SRH and health status. Nevertheless, Case et al. (2002) show a positive correlation between children’s poor health outcomes and a wide variety of chronic health problems, conditional on parental income. This includes conditions such as epilepsy, kidney disease, asthma, diabetes, and mental retardation, which are likely to be more objective health measures.

A related concern with SRH given our focus on children is that the latter are not responding to the survey themselves, and thus, “self-rated health” in our study of children should be understood as health reported by the household respondent, typically a parent or adult caregiver. Nevertheless, ancillary studies using data on parent’s and children’s reports of children’s health indicate that individual measures of health status are not statistically different based on whether the report is coming from a child or a parent (Case et al. 2002). Thus, we follow the practice of denoting health as “self-rated” although an adult respondent is reporting health for children and argue that this is likely to be close to the health status of the children were they responding for themselves.

Another concern regarding SRH in the current context is that the immigrant population may use a different set of factors to assess health than the native born population. For instance, Finch et al. (2002) show that the association between SRH and subsequent mortality is mediated by acculturation measures such as years in the USA. A related concern is that differences between the SRH of immigrants and natives may actually reflect language of interview, as prior studies have documented discrepancies in the translation of the SRH categories that may result in a downward-bias in SRH among Latinos (Bzostek et al. 2007; Viruell-Fuentes et al. 2011; Pastor et al. 2015). However, since the focus of our paper is ethnic attrition, a phenomenon that we will show is primarily observed at higher-order immigrant generations born in the USA, systemic cultural differences in SRH reporting are much less likely to be salient factors explaining our results. Similarly, studies show that the overwhelming majority of US-born Latinos are proficient in English (Krogstad et al. 2015; Hakimzadeh and Cohn 2007), and thus, Spanish translation of SRH is also not likely to play a major role underlying our findings.

Nevertheless, we can address the above concerns with SRH using a similar approach to Case et al. (2002). In supplementary analysis reported in Appendix Table 8, we use the National Health Interview Survey (NHIS) to regress an indicator for poor/fair self-rated health on a wide range of health outcomes available in that data set. Thus, we can confirm that poor self-rated health is indeed highly correlated with more objective measures of health specific to children, including, but not limited to, having a chronic condition, developmental delay, learning disorder, missed school days, allergy, asthma, or other health limitation, even after controlling for language of interview.6 Ideally, Table 8 would estimate the correlation between SRH and health conditions for ethnic attritors and non-attritors within each generation, defined using grandparents’ place of birth, but that is not possible because that information is not available in the NHIS. However, the positive correlation between poor health status and other poor health indicators among children clearly holds for 1st- and higher-order generations of Mexican immigrants, as well as non-Hispanic whites, suggesting that our findings on poor self-rated health are not simply an artifact of cultural factors which immigrants may use to grade their health status and that of their children.7 Based on descriptive statistics not reported here, we can also confirm that the extent of ethnic attrition found in the NHIS sample is similar to that found in the CPS sample used in the main results section below. This suggests that the pattern of correlation between ethnic attrition and health outcomes is likely to be similar across the two data sets as well.

In addition to this supplementary evidence showing the link between SRH and objective health outcomes, Antman, Duncan and Trejo (2016) use the NHIS to show that ethnic attritors, defined as children not identified as Mexican American despite having at least one parent identified as Mexican American, are closer to non-Hispanic whites on several measures of health, including low birth weight, obesity, and having an acute health condition or activity limitation. Ethnic attritors also more closely resemble non-Hispanic whites in terms of self-rated health. This is further evidence suggesting that self-rated health is likely to correlate well with more objective health measures for the population we focus on in this paper.

While the NHIS affords the luxury of a broader set of health outcomes, the measure of ethnic attrition available in that data set is limited by parental self-identification. This puts the NHIS at a significant disadvantage to the CPS used in the body of this paper, which allows us to leverage information on parents’ place of birth as described below. Nevertheless, the supplementary evidence using the NHIS reviewed above suggest that the poor SRH outcome available in the CPS, though not ideal, can be viewed as a reasonable proxy for true poor health status in the population under study.

2.2. Identifying Mexican generations

As mentioned above, the vast research on immigrant health and the Hispanic Health Paradox in particular has almost universally relied on self-reported ethnicity to identify the descendants of Hispanic immigrants. From this perspective, individuals are identified as Hispanic if they respond affirmatively to the Hispanic origin question which asks respondents if they are Hispanic, Spanish, or Latino.8 Respondents that answer affirmatively are then asked with which Hispanic sub-group they identify. For instance, Mexican immigrants and their descendants are identified based on whether they identify themselves as Mexican on the Hispanic origin question. Foreign-born individuals are identified as 1st-generation immigrants, US-born individuals with at least one foreign-born parent are identified as 2nd-generation immigrants, and US-born individuals with no foreign-born parents are identified as 3rd+-generation immigrants. Thus, assimilation and intergenerational progress are tracked across generations of individuals who identify as Mexican, ignoring the possibility that some individuals with Mexican ancestry may not identify themselves as Mexican.

To make these notions concrete, Table 1 presents the percentage of 20–64-year-old adults reported to have poor health status as they might be presented in the literature relying on measures of ethnic self-identification. Comparing the health outcomes of 3rd+-generation non-Hispanic whites (column 4) with 3rd+-generation self-identified Mexicans (column 3) illustrates the perceived problem of incomplete intergenerational integration: only 9.31% of 3rd+-generation non-Hispanic white men are in poor health while the analogous statistic for 3rd+-generation Mexican men is 12.18. Similarly, comparing the health outcomes of 1st-generation Mexican immigrants (column 1) with 3rd+-generation self-identified Mexicans (column 3) illustrates the perceived puzzle of 1st-generation health advantage: only 9.57% of 1st-generation Mexican male immigrants are found to be in poor health while 12.18% of 3rd+-generation Mexican men are in poor health. Importantly, both of these gaps hinge on unbiased estimation of the health status of 3rd+-generation men. This paper investigates the extent to which selective ethnic attrition may skew those estimates, thus biasing the comparison of health outcomes across generations and between ethnic groups. While a higher percentage of women report poor health status, similar comparisons can be made for Mexican women across generations and between self-identified ethnic groups, though in some cases, the magnitude of the gaps are substantially smaller.

Table 1.

Percentage of adults aged 20–64 reporting poor health status, by Mexican generation

Mexicans
3rd+-Generation whites
1st Generation 2nd Generation 3rd+ Generation

Men in poor health 9.57 9.16 12.18 9.31
(0.16) (0.29) (0.27) (0.05)
Sample size 32,859 9682 14,258 328,363
Women in poor health 12.60 10.79 13.94 10.19
(0.19) (0.30) (0.27) (0.05)
Sample size 29,720 10,613 16,157 345,826

Source: 1996–2017 CPS March supplement data. Standard errors are shown in parentheses. The sample includes men and women ages 20–64. Mexicans are identified by the Hispanic origin question. Third+-generation whites are non-Hispanic white only

A typical question raised in the literature on immigrant health is whether these disparities can be explained by not only differences in observable characteristics—at a minimum age, but also SES indicators such as educational attainment. Importantly, the regressions that investigate this question should not be interpreted as causal, as they only indicate whether the correlations between immigrant generation, ethnicity, and health persist after accounting for these important differences. We reproduce this style of regressions in Table 2, where the dependent variable is an indicator for poor or fair health, and the sample includes 1st-, 2nd-, and 3rd+-generation Mexicans as well as 3rd+-generation non-Hispanic whites (the reference group). As the latter are mutually exclusive and exhaustive categories, the estimates in columns (1) and (4) generate the raw differences across groups and can be used to reproduce the summary statistics from Table 1. Thus, we can confirm that 3rd+-generation Mexican men are 2.87 percentage points more likely to be in poor health than 3rd+-generation non-Hispanic white men, an estimate that is statistically significant at the 1% level. Columns (2) and (5) add in basic controls such as age, age squared, and a set of dummy variables for survey year and location. Additionally, columns (3) and (6) follow the literature by controlling for years of education as a measure of SES. Here we find that 3rd+-generation Mexican men are 2.20 percentage points more likely to be in poor health than 3rd+-generation non-Hispanic white men, thus indicating that this gap cannot be fully explained by differences in observable characteristics. At the same time, 3rd+-generation Mexican men are 8.11 percentage points (.0220 – .0591) more likely to be in poor health than 1st-generation Mexican male immigrants, suggesting that the puzzle of 1st-generation health advantage is only exacerbated by accounting for differences in SES.

Table 2.

Poor health regressions for Mexican generations relative to 3rd+-generation whites, by gender

Men
Women
(1) (2) (3) (4) (5) (6)

1st-generation Mexican .0026 .0250*** −.0591*** .0240*** .0411*** −.0525***
(.0017) (.0018) (.0020) (.0019) (.0020) (.0022)
2nd-generation Mexican −.0015 .0316*** .0069* .0059* .0367*** .0092**
(.0030) (.0031) (.0031) (.0030) (.0031) (.0031)
3rd+-generation Mexican .0287*** .0459*** .0220*** .0375*** .0505*** .0228***
(.0025) (.0026) (.0026) (.0025) (.0026) (.0026)
Years of education −.0186*** −.0206***
(.0002) (.0002)
Basic controls No Yes Yes No Yes Yes
Average poor health .0943 .0943 .0943 .1054 .1054 .1054
Sample size 385,162 385,162 385,162 402,316 402,316 402,316

Source: 1996–2017 CPS March supplement data. Standard errors are shown in parentheses. The sample includes 1st-, 2nd-, and 3rd+-generation Mexicans and 3rd+-generation non-Hispanic whites (the reference group), ages 20–64. Mexicans are identified by the Hispanic origin question. Each column reports estimates from a separate ordinary least squares regression of the form: Hi=βIi+Xiπ+εi, where Hi=1 if individual i has poor/fail health, and Ii is a vector including the following dummy variables: 1st-generation Mexican, 2nd-generation Mexican, and 3rd+-generation Mexican. Basic controls include age, age squared, and dummy variables for survey year, division of the country, individual states NY, NJ, IL, FL, TX, NM, AZ, CA, and urban area

Interestingly, the puzzle of 1st-generation advantage also rules out one possible explanation for the gap between later-generation Mexicans and non-Hispanic whites: 3rd+-generation Mexicans are not in worse health than 3rd+-generation non-Hispanic whites because unhealthy Mexicans arrive in the USA. In fact, after controlling for socioeconomic status, we see that 1st-generation Mexicans immigrants are actually less likely to be in poor health than non-Hispanic whites (point estimate – .0591 for men). More importantly, the evidence presented here suggests that something is happening across immigrant generations in the USA that is making the descendants of Mexican immigrants less healthy or at least appear to be less healthy. The remainder of this paper investigates the extent to which selective ethnic attrition skews our perception of that phenomenon.

Throughout the paper, we use the following definitions to classify immigrant generation based on more objective information on place of birth of parents and grandparents.9 The 1.5-generation Mexicans are children born in Mexico. Second-generation Mexicans are US-born and have at least one parent born in Mexico. Third-generation Mexicans are US-born, have two US-born parents, and have at least one grandparent born in Mexico.10 Finally, 4th+-generation Mexicans are US-born, have two US-born parents, have no grandparents born in Mexico, and have at least one parent who identifies as Mexican on the Hispanic origin question.11 As a comparison, 4th+-generation whites have no parents or grandparents who are foreign-born, and themselves and both parents are identified as non-Hispanic on the Hispanic origin question and white only on the race question.

The CPS provides information on the country of birth of an adult respondent’s parents, but not of his or her grandparents. Unfortunately, this makes it impossible for us to construct an objective measure of 3rd- and 4th+-generation Mexican adults. However, by linking children to their parents’ records, we are able to identify the country of birth of a child’s grandparents. For this reason, the analysis sample in the remainder of the paper is limited to children ages 17 and younger living in intact families at the time of the survey, since information on parents and grandparents on both sides of an individual’s family are only available for this group.12 While our focus on children is necessary due to data limitations, it also means this study makes another important contribution to the literature on immigrant advantage and intergenerational health which typically focuses on adult health outcomes.13 However, the fact that the immigrant children we observe came to the USA at an early age suggests they are likely to display health outcomes much closer to their US-born counterparts compared with immigrants who arrived as adults. This caveat is further complicated by the fact that children are generally less likely than adults to be in poor health. As a result, it is not obvious that we should expect to see the same story of declining immigrant advantage which we observed for adults in Table 1. Nevertheless, we can still examine the role of selective ethnic attrition in biasing estimates of health status for US-born Mexicans. As we presume any health differentials will be compounded over the life course, we expect that any biases observed here are likely to be understated compared with estimates we might obtain if it were possible to observe ethnic attrition in a sample of 3rd+-generation adults.14

3. Results

Table 3 begins by documenting the extent of ethnic attrition of the children in our sample and linking this with whether their parents intermarried, that is whether they have Mexican ethnicity on one or both sides of the family. The definition of Mexican ethnicity on one side of the family is consistent across all generations. For example, a child has Mexican ethnicity on his father’s side of the family if any of the following are true: (1) the child has a paternal grandparent who was born in Mexico; (2) the child’s father was born in Mexico; or (3) the child’s father self-identifies as Mexican.

Table 3.

Mexican identification of children ages 17 and younger, by source of Mexican ethnicity

1.5 Generation 2nd Generation 3rd Generation 4th+ Generation

Percent with Mexican ethnicity on:
 Both sides of the family 98.3 89.9 56.0 38.7
 One side of the family 1.3 10.1 44.0 61.3
 Father’s side only 0.3 5.4 22.9 30.7
 Mother’s side only 1.0 4.7 21.1 30.7
 Either side of the family 99.6 100.0 100.0 100.0
Percent identified as Mexican with:
 Mexican on both sides of the family 98.00 97.43 96.96 98.59
(.19) (.10) (.30) (.18)
 Mexican on one side of the family 85.71 67.92 61.46 63.32
(4.21) (.86) (.96) (.59)
  Mexican on father’s side only 75.00 66.58 63.11 61.64
(11.18) (1.19) (1.32) (.84)
  Mexican on mother’s side only 88.89 69.47 59.66 65.00
(4.32) (1.25) (1.40) (.83)
All 97.50 94.45 81.35 76.96
(.21) (.13) (.51) (.40)
Sample size 5328 29,185 5867 10,830

Source: 1996–2017 CPS March supplement data. Standard errors are shown in parentheses. The samples include US- and Mexican-born children ages 17 and below who live in intact families. The 1.5-generation Mexicans are children born in Mexico. Second-generation Mexicans are US-born and have at least one parent born in Mexico. Third-generation Mexicans are US-born, have two US-born parents, and at least one grandparent born in Mexico. Fourth+-generation Mexicans are US-born, have two US-born parents, have no grandparents born in Mexico, and at least one parent who identifies as Mexican on the Hispanic origin question

Following the conventional definition, children born in Mexico are considered 1.5 generation regardless of where their parents or grandparents were born. In our sample, there are 21 1.5-generation children who do not have any Mexican parents or grandparents. Excluding these children from the analysis does not change any of the results

As is seen from Table 3, one important empirical result is that Mexican identification is strongly linked with intermarriage. Across all generations, Mexican identification is almost universal (97 to 99%) among children of endogamous marriages, that is, those children with Mexican ethnicity on both sides of the family. In contrast, with the exception of “1.5-generation” Mexicans who are immigrants themselves, rates of ethnic identification are far lower (61 to 68%) for individuals with Mexican ethnicity on only one side of the family, that is, children of intermarriages between one Mexican and one non-Mexican. These patterns are also fairly consistent regardless of whether the Mexican ethnicity is on the father’s or mother’s side of the family.

It follows that the extent of ethnic attrition across generations will be closely linked with the extent of intermarriage across generations. This pattern is confirmed in Table 3, which mirrors findings elsewhere in the literature highlighting intermarriage as the primary source of ethnic attrition (Duncan and Trejo 2011, 2016, 2017; Antman et al. 2016). For instance, a very small share of 2nd-generation children have intermarried parents (10.1%), that is, Mexican ethnicity on only one side of the family. Thus, it comes as no surprise that there is very little ethnic attrition at the 2nd generation, and in fact, 94.45% of 2nd-generation children identify as Mexican. This is similar to the rate of Mexican identification among 1.5-generation children (97.50%), who we emphasize are immigrants themselves, and who are almost exclusively the children of endogamous marriages (98.3%). In contrast, rates of intermarriage are quite substantial for the parents of 3rd- and 4th+-generation children (44% and 61.3%, respectively). Consequently, rates of Mexican identification are far lower for the latter groups (81.35% and 76.96%, respectively). This implies that substantial shares of 3rd- and 4th+-generation children are not identified as Mexican (18.65% and 23.04%, respectively).15 Since ethnic attrition is a phenomenon primarily occurring at the 3rd and higher generations, in the remainder of the paper, we limit our analysis of ethnic attrition to these groups.

Thus far, we have established that intermarriage rates are substantial at the 3rd and higher generations and ethnic attrition is closely linked with parental intermarriage.16 We now proceed in connecting parental intermarriage with parental health. This is done in Table 4, which shows the percent of parents in poor health based on whether children have two Mexican parents (Mexican on both sides of the family) or one Mexican and one non-Mexican parent (Mexican on one side of the family). With regard to intermarriage, it is noteworthy that both fathers and mothers in intermarriages are less likely to be in poor health. For example, about 6% of fathers who are intermarried report poor SRH, whereas 8.55 to 9.76% of fathers who are not intermarried report poor SRH at the 3rd and 4th+ generations, respectively.17 Thus, intermarriage between Mexicans and non-Mexicans appears to be selective on health.18

Table 4.

Poor health status of parents of children ages 17 and younger, by source of Mexican ethnicity

3rd Generation 4th+ Generation

Percent of fathers reporting poor health status:
 Mexican on both sides of the family 8.55 9.76
(.49) (.46)
 Mexican on one side of the family 5.82 6.08
(.46) (.29)
 All 7.35 7.51
(.34) (.25)
Percent of mothers reporting poor health status:
 Mexican on both sides of the family 6.84 7.35
(.44) (.40)
 Mexican on one side of the family 5.93 6.72
(.47) (.31)
 All 6.44 6.96
(.32) (.24)

Source: 1996–2017 CPS March supplement data. Standard errors are shown in parentheses. The samples include US-born children ages 17 and below who live in intact families. Third-generation Mexicans are US-born, have two US-born parents, and at least one grandparent born in Mexico. Fourth+-generation Mexicans are US-born, have two US-born parents, have no grandparents born in Mexico, and at least one parent who identifies as Mexican on the Hispanic origin question

Table 5 completes the picture by linking parental health directly with children’s ethnic identification. Since previous tables have already established the close connection between ethnic attrition and intermarriage, it should come as no surprise that the pattern of results look strikingly similar to Table 4. For the most part, health status is better for parents of those children who are not identified as Mexican (parents of ethnic attritors), compared with the parents of children that are identified as Mexican (parents of non-attritors). For instance, 8.29% of fathers of 4th+-generation children identified as Mexican report being in poor health compared with only 4.89% of fathers of 4th+-generation children not identified as Mexican. The same pattern is apparent for fathers of 3rd-generation children, albeit with a smaller gap (7.75% versus 5.58%). Assuming an intergenerational correlation in health status, this would suggest that ethnic attritors themselves are also likely to be in better health compared with non-attritors.

Table 5.

Poor health status of parents of children ages 17 and younger, by child’s Mexican identification

3rd Generation 4th+ Generation

Percent of fathers reporting poor health status:
 Child identified as Mexican 7.75 8.29
(.39) (.30)
 Child not identified as Mexican 5.58 4.89
(.69) (.43)
 All 7.35 7.51
(.34) (.25)
Percent of mothers reporting poor health status:
 Child identified as Mexican 6.39 7.13
(.35) (.28)
 Child not identified as Mexican 6.67 6.41
(.75) (.49)
 All 6.44 6.96
(.32) (.24)

Source: 1996–2017 CPS March supplement data. Standard errors are shown in parentheses. The samples include US-born children ages 17 and below who live in intact families. Third-generation Mexicans are US-born, have two US-born parents, and at least one grandparent born in Mexico. Fourth+-generation Mexicans are US-born, have two US-born parents, have no grandparents born in Mexico, and at least one parent who identifies as Mexican on the Hispanic origin question

Table 6 addresses this question directly by documenting the percentage of children reporting poor health status by generation and ethnic identification. Importantly, we note that sample sizes are small and differences in point estimates are also small given the nature of the SRH outcome and the focus on children in our study. Thus, we do not aim to find statistically significant differences among our sample of children, only suggestive indications of differences in the broader population. With these caveats in mind, and as expected given the results from above, we find that the patterns displayed in Table 6 are consistent with the results from above: ethnic attrition is selective on the health of children, with the degree of selectivity varying by generation. Third-generation Mexican children who do not identify as Mexican are somewhat less likely to report being in poor health compared with 3rd-generation Mexican children who identify as Mexican (1.46% versus 1.74%, respectively). Given this relatively small difference, the percent of 3rd-generation Mexicans estimated to be in poor health which includes all 3rd-generation Mexicans based on grandparents’ place of birth (1.69%) is close to the estimate which relies on Mexican self-identification (1.74%). For the 4th+ generation, however, the gap in poor health status is somewhat larger for ethnic attritors versus non-attritors (1.44% versus 1.82%, respectively). As a result, the overall percentage of 4th+-generation Mexicans reported to be in poor health (1.74%) is somewhat lower than the estimate based on self-identification (1.82%).19 In short, as ethnic attritors are positively selected on health, conventional estimates of health outcomes for higher-order immigrant generations make them appear to be in worse health than they actually are.

Table 6.

Percentage of children ages 17 and younger reporting poor health status, by Mexican generation

Mexicans
1.5 Generation 2nd Generation 3rd Generation identified as Mexican 4th+ Generation identified as Mexican 4th+ Generation white


Yes No All Yes No All

Poor health (%) 2.59 2.52 1.74 1.46 1.69 1.82 1.44 1.74 1.24
(.22) (.09) (.19) (.36) (.17) (.15) (.24) (.13) (.02)
Sample size 5328 29,185 4773 1094 5867 8335 2495 10,830 211,699

Source: 1996–2017 CPS March supplement data. Standard errors are shown in parentheses. The samples include US- and Mexican-born children ages 17 and below who live in intact families. The 1.5-generation Mexicans are children born in Mexico. Second-generation Mexicans are US-born and have at least one parent born in Mexico. Third-generation Mexicans are US-born, have two US-born parents, and at least one grandparent born in Mexico. Fourth+-generation Mexicans are US-born, have two US-born parents, have no grandparents born in Mexico, and at least one parent who identifies as Mexican on the Hispanic origin question. Fourth+-generation whites have no parents or grandparents who are foreign-born, and themselves and both parents are identified as non-Hispanic on the Hispanic origin question and white only on the race question

Table 6 also provides insight into how selective ethnic attrition might bias standard comparisons of health outcomes across generations of Mexican Americans and between Mexican Americans and non-Hispanic whites.20 Before discussing these results, it should be emphasized that the 1.5-generation immigrants in our sample came to the USA as children and are thus likely to display health outcomes much closer to those of US-born children than, say, adults who were born in Mexico and migrated as adults. This caveat is compounded by the fact that in general children are less likely to be in poor health relative to adults and thus health disparities are likely to be understated for this group. As a result, for child self-rated health, as with other outcomes in the literature on immigrant health (Riosmena et al. 2013), we do not find a clear story of immigrant advantage.21 However, ultimately, our interest is not in documenting immigrant advantage and assimilation gaps in the SRH of children specifically, but rather in using the sample of children to estimate the extent of ethnic attrition and the degree to which it is selective on health. While there are no studies to our knowledge that indicate how child SRH translates into adult SRH, if we reasonably presume that they are correlated, we can use the Table 6 results to get a rough estimate of the magnitude of the effect ethnic attrition has on the adult patterns seen in Table 1. This may be a lower bound estimate if, for example, a parent’s adult SRH is transmitted to her child, but this is more likely to show up in adult SRH versus child SRH. To the extent that additional ethnic attrition occurs as children transition into adulthood, we might also regard this as an underestimate.

Thus, we use the results from Table 6 to obtain a rough approximation of the size of the bias due to ethnic attrition which could affect estimates of 1st-generation advantage and long-term integration for the adult population documented in Table 1. In particular, estimates for the 4th+ generation from Table 6 suggest that ethnic attrition is associated with an increased likelihood of reporting poor health status by 4.60% [(1.82–1.74)/1.74]. If the same effect were to apply to the 3rd+-generation adults in Table 1, the corrected percentage of respondents reporting poor health status would be 11.64% (12.18/1.0460). This implies that the gap between 3rd+ and 1st-generation Mexicans is about 21% smaller (11.64–9.57 versus 12.18–9.57) than suggested by conventional estimates based on subjective measures of ethnic identification. Similar analysis implies that using more objective measures of ethnic identification would yield a reduction of about 19% (11.64–9.31 versus 12.18–9.31) in the health gap between 3rd+-generation Mexicans and 3rd+-generation whites. In other words, both the 1st-generation health advantage and the health disadvantage of later-generation Mexicans (relative to non-Hispanic whites) are substantially overstated. This bias stems from the relatively large difference in health status between ethnic attritors and non-attritors at the 4th+ generation.

Table 7 examines these health differentials further by investigating whether they survive after accounting for observable differences such as gender, age, and SES.22 To do this, we first estimate a regression on the sample of all groups noted in Table 6, where the dependent variable is an indicator for poor health status and the right-hand side includes a mutually exclusive and exhaustive set of dummy variables accounting for all of the categories featured at the top of Table 6, with the exception of 4th+-generation non-Hispanic whites (the reference group). Thus, the estimates in column (1) can be used to reproduce the differences in health outcomes across the columns from Table 6 and confirm that ethnic attritors at the 4th+ generation have health outcomes similar to those of 4th+-generation non-Hispanic whites (coefficient estimate 0.0020). Column (2) adds basic controls for gender, age, year, and location, with little change in the results: self-identified Mexicans at the 4th+ generation are about 0.55 percentage points more likely to be in poor health relative to 4th+-generation non-Hispanic whites. However, this health differential is cut by more than half once we add controls for socioeconomic status (column 3), dropping the coefficient estimate on 4th+-generation self-identified Mexicans to a statistically insignificant 0.0026. Once we control for parental health characteristics (column 4), the difference between self-identified Mexicans and non-Hispanic whites disappears almost completely (coefficient estimate 0.0014). More importantly, the health differential between ethnic attritors and non-attritors at the 4th+ generation is not statistically significant after controlling for parental characteristics. This suggests that the relationship between health and ethnic attrition is driven by parental characteristics—the health differential between ethnic attritors and non-attritors effectively disappears once we control for those parental characteristics determining ethnic attrition.

Table 7.

Child poor health regressions for Mexican generations relative to 4th+-generation whites

(1) (2) (3) (4)

1.5-generation Mexican .0135*** .0132*** .0019 .0043*
(.0017) (.0017) (.0018) (.0018)
2nd-generation Mexican .0128*** .0135*** .0045*** .0052***
(.0007) (.0009) (.0010) (.0010)
3rd-generation Mexican:
 Identified as Mexican .0050** .0052** .0025 .0016
(.0017) (.0018) (.0018) (.0018)
 Not identified as Mexican .0022 .0020 .0006 −.0003
(.0036) (.0036) (.0036) (.0036)
4th+-generation Mexican:
 Identified as Mexican .0058*** .0055*** .0026 .0014
(.0013) (.0014) (.0014) (.0014)
 Not identified as Mexican .0020 .0017 .0000 −.0001
(.0024) (.0024) (.0024) (.0024)
Basic controls No Yes Yes Yes
SES No No Yes Yes
Parental health No No No Yes
Average poor health .0144 .0144 .0144 .0144
Sample size 262,909 262,909 262,909 262,909

Source: 1996–2017 CPS March supplement data. Standard errors are shown in parentheses. The samples include US- and Mexican-born children ages 17 and below who live in intact families. The 1.5-generation Mexicans are children born in Mexico. Second-generation Mexicans are US-born and have at least one parent born in Mexico. Third-generation Mexicans are US-born, have two US-born parents, and at least one grandparent born in Mexico. Fourth+-generation Mexicans are US-born, have two US-born parents, have no grandparents born in Mexico, and at least one parent who identifies as Mexican on the Hispanic origin question. Fourth+-generation whites have no parents or grandparents who are foreign-born, and themselves and both parents are identified as non-Hispanic on the Hispanic origin question and white only on the race question. Fourth+-generation whites are the reference group. Each column reports estimates from a separate ordinary least squares regression of the form: Hi=βIi+Xiπ+εi, where Hi=1 if child i has poor/fail health, and Ii is a vector including the following dummy variables: 1.5-generation Mexican, 2nd-generation Mexican, 3rd-generation Mexican identified as Mexican, 3rd-generation Mexican not identified as Mexican, 4th+-generation Mexican identified as Mexican, and 4th+-generation Mexican not identified as Mexican. Controls (Xi), where indicated, are as follows. Basic controls include age, age squared, and dummy variables for gender, survey year, division of the country, individual states NY, NJ, IL, FL, TX, NM, AZ, and CA, and urban area. SES controls include controls for mother’s and father’s years of education and employment status. Parental health controls include dummy variables for mother’s and father’s self-reported health status

4. Conclusion

This paper has investigated the role of ethnic attrition in biasing estimates of the health status of US-born Mexicans. We find that at higher-order immigrant generations, ethnic attritors display better health outcomes than non-attritors and closer to those of non-Hispanic whites. This implies that estimates of health status which rely on ethnic self-identification make Mexican Americans appear to be in worse health than they actually are. As a result, standard evidence of incomplete integration, showing US-born Mexicans in significantly worse health than non-Hispanic whites, is significantly overstated. Similarly, these results suggest that failing to account for the relatively better health of ethnic attritors is likely to overstate any estimated 1st-generation health advantage which relies on self-reported ethnic identification.

Additionally, we find that once we control for parental health and SES, the health differential by ethnic attrition disappears, reinforcing the view that ethnic attrition is driven by parental characteristics. We also show that intermarried individuals are likely to be in better health and are more likely to produce children that are not identified as Mexican who are also in better health. Together, this collection of results suggests that selective ethnic attrition, driven by parental characteristics that are culled through a process of selective intermarriage, biases our estimates of the health of US-born Mexicans and thus skews our perceptions of immigrant advantage and intergenerational integration. This is consistent with earlier work (Antman, Duncan and Trejo 2016) showing similar results applied to a broader range of health outcomes, but relying on a more limited measure of ethnic attrition across immigrant generations. Future work should investigate other measures of well-being used in the cultural assimilation literature, such as life satisfaction (Angelini et al. 2015), to assess the impacts of ethnic attrition on a wider set of outcomes.

While our estimates suggest that ethnic attrition is an important phenomenon overlooked by research studies that rely solely on subjective measures of lineage, an important caveat of this study is that data limitations inherent in the CPS restrict our analysis to self-rated health status on a sample of children. While we have shown that SRH is correlated with a wide array of health outcomes, future data collection efforts should increase the availability of objective variables documenting ancestral origins such as antecedents’ places of birth and link these with a wider set of objective health outcomes and health behaviors, thus facilitating research in this area. Despite these limitations, we have argued that our estimates of the magnitude of ethnic attrition and resulting biases are likely to be understated. Since the analysis here is limited to children, it is likely that our estimates of the impact of ethnic attrition on measured health disparities are likely to be underestimated because ethnic attrition rates are likely to be higher than what we observe here just as differences in health outcomes are likely to become all the more salient as children transition to adulthood. Consequently, we should expect the bias due to ethnic attrition that is present in current estimates of health differentials across immigrant generations and ethnic groups to be even larger in magnitude than what is suggested here.

Acknowledgments

We thank Brian Cadena, Terra McKinnish, Joseph P. Price, Fernando Riosmena, and conference participants at the Society of Labor Economists meeting and the Western Economics Association International meeting for their feedback. This work also benefited from the comments of four anonymous referees and the editor, Klaus F. Zimmermann

Appendix

Table 8.

The relationship between health conditions and poor/fair health among 0–17-year-old children, by Mexican generation

1.5-Generation Mexican 2nd-Generation Mexican 3rd+-Generation Mexican 3rd+-Generation whites




Sample size Fraction w/ cond. β^ Sample size Fraction w/ cond. β^ Sample size Fraction w/ cond. β^ Sample size Fraction w/ cond. β^

Chronic condition 5946 .0254 .1151*** 37,870 .0392 .1392*** 13,729 .0573 .0920*** 105,627 .0669 .0601***
(.0281) (.0096) (.0108) (.0030)
Health limitation 5946 .0262 .1107*** 37,870 .0400 .1358*** 13,729 .0584 .0914*** 105,627 .0679 .0594***
(.0273) (.0094) (.0107) (.0029)
Health condition 5946 .103 .0306*** 37,870 .127 .0401*** 13,729 .182 .0157*** 105,627 .205 .0126***
(.0092) (.0035) (.0034) (.0010)
Asthma1 2356 .0463 .0548** 15,629 .0807 .0774*** 6202 .121 .0306*** 51,610 .107 .0203***
(.0255) (.0085) (.0077) (.0023)
Allergy1 2354 .129 .0332** 15,611 .162 .0463*** 6190 .256 .0135*** 51.491 .276 .0136***
(.0133) (.0051) (.0041) (.0012)
Ear infection1 2358 .0310 .0316 15,626 .0604 .0609*** 6207 .0657 .0428*** 51,638 .0656 .0211***
(.0271) (.0091) (.0109) (.0029)
Developmental delay1 2357 .0132 .1051* 15,625 .0188 .1599*** 6207 .0350 .0799*** 51,627 .0372 .0738***
(.0603) (.0225) (.0195) (.0062)
Chicken pox1 2331 .417 .0073 15,558 .207 −.0019 6172 .228 .0032 51,338 .289 .0014
(.0072) (.0042) (.0047) (.0013)
Low birth weight1 1932 .138 .0078 14,353 .0798 .0208*** 5902 .0749 .0046 49.885 .0700 .0115***
(.0113) (.0064) (.0065) (.0024)
ADHD1 2323 .0125 −.0237*** 13.192 .0202 .1109*** 5017 .0439 .0211 44,073 .0660 .0215***
(.0074) (.0215) (.0134) (.0032)
Learning disorder1 2281 .0285 .0808** 12,034 .0446 .0959*** 4494 .0679 .0530*** 40,685 .0665 .0435***
(.0385) (.0143) (.0142) (.0043)
Missed school days1,2 485 2.106 .0037** 9761 2.531 .0066*** 3603 3.447 .0027*** 34,246 3.538 .0036***
(.0016) (.0010) (.0010) (.0003)
Obese1,2 485 .167 .0326 2157 .226 .0105 695 .153 .0231 7653 .106 .0204***
(.0245) (.0104) (.0199) (.0064)

Source: 2000–2015 National Health Interview Survey (NHIS). The sample includes US- and Mexican-born children ages 0–17 living with both biological parents. Sample further limited to the following:

1

sample children and

2

ages 12–17 in the sample years 2008–2014.

The 1.5-generation Mexican children are foreign-born children with at least one foreign-born parent. Second-generation Mexican children are US-born children with at least one foreign-born parent. Third+-generation Mexican children are US-born with two US-born parents. Each β^ is from a separate ordinary least squares regression of the form: Hi=βCi+Xiπ+εi, where Hi=1 if child i has poor/fail health, and Ci=1 if child i has the indicated health condition. Controls Xi include child’s gender, child’s integer age fixed effects, mother’s and father’s age (quadratic), mother and father’s education fixed effects, 13 family income fixed effects, an indicator variable equal to one if the interview was conducted in Spanish, region fixed effects, and year fixed effects

Footnotes

Compliance with ethical standards

Conflict of interest The authors declare that they have no conflict of interest.

1

Additional work in this literature examines whether any health advantage erodes over time within the 1st immigrant generation (e.g. Acevedo-Garcia et al. 2010). Since our study makes comparisons across immigrant generations, however, we do not focus on years since migration.

2

With regard to death rates in particular, one explanation sometimes offered for the paradox vis-à-vis non-Hispanic whites stems from differences in reporting of ethnic identity by next-of-kin on death certificates relative to self-reporting of ethnicity on national surveys (Rosenberg et al. 1999). While investigations of this explanation do not constitute an extensive portion of the literature, they do hint at problems inherent in the use of subjective measures of ethnic identity more broadly.

3

This is similar to the mechanism suggested by Giuntella (2016) to be the root cause for the decline in birth weight observed across immigrant generations, which he attributes to the intermarriage of non-Hispanic whites with 2nd-generation Hispanic immigrants who are then more likely to adopt risky behaviors such as smoking during pregnancy.

4

Duncan and Trejo (2016, 2017) investigate ethnic attrition among other Hispanic groups and find similar patterns of educational selectivity across all Hispanic origin groups, but precise estimates of the magnitude of the implied biases are thwarted by the very small sample sizes of the non-Mexican groups. While this evidence suggests that health selectivity patterns are likely to be similar across Hispanic-origin groups as well, in practice, the small sample sizes associated with other Hispanic immigrants in the CPS limit our ability for precise comparisons with the Mexican-origin group.

5

We follow the shorthand, common in the literature, of describing this outcome as a poor health indicator, although, as noted above, it also includes those reported to be in fair health. Typically, the literature constructs the SRH measure as a binary measure of health status, reflecting the fact that indicators for poor or fair health in particular are good predictors of subsequent health outcomes such as mortality (Wolinsky and Johnson 1992).

6

One important limitation of these alternative health measures, as with almost all health outcomes available in large-scale data sets, is that they are self-reported and thus potentially subject to measurement error biases. Nevertheless, we argue that these are likely to be smaller in magnitude than those associated with SRH, especially for those measures that are closely linked with an objective health condition (e.g. low birth weight). If one assumes that these measurement error biases reflect measures of acculturation to US norms, we might also expect them to result in understated differences between higher-order immigrants and non-Hispanic whites.

7

While Table 8 shows that low birth weight, which has been linked with smoking during pregnancy for higher-order immigrants (Giuntella 2016, 2017), displays a statistically significant correlation with SRH, it should be noted that the rest of the health outcomes in Table 8 cannot be strictly linked with risky behaviors. This limits our capacity to investigate the extent to which cultural assimilation and intermarriage can be linked with poor health outcomes for higher-order immigrant generations by way of risky health behaviors, as has been explored elsewhere in the literature (Giuntella 2016, 2017; Edwards and Goldstein 2018). Nevertheless, as the focus of this paper is on documenting the impact of ethnic attrition on health outcomes using the CPS, we return in the body of the paper to focusing on SRH and leave analysis of the link between ethnic attrition, risky behaviors, and resultant health outcomes for future research.

8

It should be noted that surveys typically ask one respondent to report the racial and ethnic identities of all individuals within the household. We follow the common practice in the literature of describing this as “self-reported” identity, though, to be clear, this may be the identity of the individual as viewed by close family or household members.

9

While we argue that antecedents’ place of birth is a more objective measure of ancestry than self-reported ethnic identity, it should be noted that place of birth is also self-reported and thus may also be subject to reporting biases. For instance, children of intermarried parents may be less likely to have Mexican grandparents listed in the survey and thus appear to the researcher to be non-Hispanic white. As we expect, these additional levels of unobserved ethnic attrition will operate in the same direction as the patterns of ethnic attrition we do observe; any biases resulting from failures to report Mexican ancestry make it less likely that we observe differences between Mexicans and non-Hispanic whites. As a result, our estimates of the impact of ethnic attrition on measured health disparities are likely to be understated.

10

We emphasize that this is the “exact” 3rd generation as opposed to 3rd generation and higher (3rd+).

11

Some might describe this as the Mexican ancestry population more generally, as their specific immigrant generation is unclear. Its reliance on self-reported ethnic identity also raises concerns that estimates of health disparities involving this group may reflect endogeneity biases associated with levels of acculturation that affect health outcomes, as well as self-reported identities, of parents and children. Nevertheless, our use of the 4th+-generation terminology and comparison group parallels the literature’s broad use of the “3rd+” generation described above and as with the latter measure will tend to underestimate ethnic attrition due to its reliance on parental self-identification. While the goal of this exercise is purely descriptive, our contribution here is to take this somewhat nebulous concept back one full generation.

12

Our focus on children also means that our notion of “self-reported” identity is actually reported by the household respondent, typically a parent or adult caregiver. We view this response as likely to be consistent with the child’s own ethnic identity at the time of the survey, since parents are important shapers of their children’s identities. In any case, we expect that parental reporting likely yields an underestimate of ethnic attrition relative to asking children their own ethnic identities as adults once they have established households separate from their parents. Duncan and Trejo (2011a, Table 9) show that children’s observed rates of Mexican identification in CPS data do not seem to vary systematically with which household member answered the CPS questionnaire (father, mother, or other household member) and with which parent provides the child’s Mexican origins (father, mother, or both).

13

Hamilton et al. (2011) provide a detailed analysis of how four common child health conditions vary by immigrant generation and race/ethnicity. They mention ethnic attrition as a potential explanation for some of the observed generational patterns, but their data do not allow them to investigate this issue empirically.

14

While it is possible, strictly speaking, to analyze the selectivity of ethnic attrition in our sample of 2nd-generation adults, ethnic attrition rates are very small for this group (less than 8%), perhaps unsurprisingly since they are the children of immigrants.

15

Further analysis, not reported here, confirms that ethnic attritors are not only attriting out of Mexican identity but also attriting out of Hispanic identity altogether. Fewer than 2% of children with Mexican ancestry identify as Hispanic, but not Mexican, at the 4th+ generation. Consequently, the results below documenting correlations with ethnic attrition out of Mexican identity are substantially similar if attrition is defined as attriting from Hispanic identity.

16

While Table 3 confirms that the primary source of ethnic attrition in our sample is intermarriage, we emphasize that the analysis that follows does not restrict attention to sources of ethnic attrition resulting from intermarriage but rather estimates the net effect of all sources of ethnic attrition on measured health disparities. Closer analysis of the determinants and implications of ethnic attrition, holding intermarriage constant, is limited by the relatively small numbers of ethnic attritors from endogamous marriages but would be an interesting avenue for future research.

17

In extended analysis not emphasized here, both the Mexican and non-Mexican spouse are likely to report better health. This is similar to the result from the assortative mating literature which finds that individuals are matched on measures of human capital such as education (Mare 1991; Pencavel 1998).

18

While the link between health and intermarriage is suggestive of a mechanism that is selective on health, we acknowledge that this process may be muddled by simultaneity in mate selection and health choices, which may in turn affect our measures of SRH that are reported after marriage has occurred. As our intent is ultimately to establish correlations between health and ethnic attrition, we leave further exploration of this mechanism to future research.

19

In additional results not presented here, we have used an alternative, more flexible, definition of 4th+ generation that includes individuals that are US-born, have two US-born parents, have no grandparents born in Mexico, and at least one parent who identifies as Hispanic on the Hispanic origin question. Those results are substantially similar to the results presented here, even after restricting the analysis to states that were historically linked with the Mexican-origin population, i.e., California, Illinois, and Texas.

20

Owing to the smaller sample sizes, we consider outcomes for girls and boys together and do not examine the selectivity of ethnic attrition at the 1.5 and 2nd generations, where rates of ethnic attrition are especially low.

21

While data limitations prevent us from comparing 1.5-generation Mexicans with the populations from which they originated, the fact that we find no clear evidence of an immigrant health advantage for the children in the sample also suggests that there are not likely to be any clear patterns of positive migrant selection on health for immigrants that arrived as children. Future research could compare child migrants with children in Mexico to investigate this directly.

22

To be consistent with the literature, the SES controls include mother’s and father’s years of education and employment status (Antecol and Bedard 2006).While the latter measure in particular is not included to estimate a causal relationship, it can be viewed as a control that may capture important differences across individuals, such as access to health care. In additional analysis not reported here, we have explored controlling for health insurance directly in analogous regressions on the population of children ages 14 and younger and found that it did not meaningfully affect the pattern of results.

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