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. 2024 Jan 26;46(3):3503–3505. doi: 10.1007/s11357-024-01085-4

Correction to: The CEPH aging cohort and biobank: a valuable collection of biological samples from exceptionally long‑lived French individuals and their offspring for longevity studies

Alexandre How‑Kit 1,2,, Mourad Sahbatou 1, Lise M Hardy 1,2, Nicolas P Tessier 1,2, Valérie Schiavon 3, Hélène Le Buanec 3, Jean‑Marc Sebaoun 4, Hélène Blanché 2,4, Jean‑François Zagury 5, Jean‑François Deleuze 1,2,4,6,
PMCID: PMC11009164  PMID: 38273141

Correction to: GeroScience

10.1007/s11357-023-01037-4

The original version of this article contained a few errors introduced during the typesetting process.

There were errors in the brackets used to delimit age intervals in the abstract and Table 2. We have corrected the age intervals to avoid any confusion.

“semi-supercentenarians ([105–110] years old)” has been corrected to “semi-supercentenarians ([105–109] years old)” in the Abstract.

“[90–100]; nonagenarians”, “[100–105]”, and “[105–110]; semi-supercentenarians” have been corrected to “[90–99]; nonagenarians”, “[100–104]”, and “[105–109]; semi-supercentenarians” in Table 2, respectively. 

New Table 2:

Table 2.

Demographic characteristics of the long-lived individuals from the CEPH Aging cohort

Age range (years) Participants (N) considering their age at inclusion Participants (N) considering their age at death Participants (N) considering their age at last known health status
All (% of women) Unrelated All (% of women) Unrelated All (% of women) Unrelated
≥ 90 1712 (75.76) 1411 1454 (76.96) 1207 1746 (75.43) 1412
[90-99] ; nonagenarians 710 (63.10) 487 347 (54.47) 234 481 (54.68) 321
≥100 ; centenarians 1002 (84.73) 996 1107 (84.01) 1063 1265 (83.32) 1212
[100-104] 930 (84.62) 927 874 (82.49) 841 984 (82.22) 944
[105-109] ; semi-supercentenarians 66 (84.85) 66 218 (90.37) 218 256 (87.89) 255
≥110 ; supercentenarians 6 (100) 6 15 (80) 15 25 (80) 25

Table 3 was incorrectly justified and had a typo, which has been corrected.

Table 3.

Quantitative epidemiologic and clinical data available for long-lived individuals from the CEPH Aging cohort

All Men Women
% of data available Mean ± SD % of data available Mean ± SD % of data available Mean ± SD
Anthropometry
  Age at inclusion (years) 100 98.9 ± 4.17 100 96.6 ± 4.57 100 99.64 ± 3.73
  Height (cm) 55.78 160.06 ± 8.65 61.77 167.94 ± 7.1 53.83 157.11 ± 7.21
  Weight (kg) 58.07 54.98 ± 12.75 62.23 65.84 ± 10.68 56.71 51.09 ± 11.08
Cardiovascular parameters
  Pulse rate (bpm) 49.94 74.23 ± 8.88 50.34 71.5 ± 8.61 49.81 75.13 ± 8.79
  Systolic blood pressure (cm Hg) 58.93 13.65 ± 1.6 58.74 13.57 ± 1.45 58.99 13.67 ± 1.64
  Diastolic blood pressure (cm Hg) 58.19 7.63 ± 0.87 58.04 7.66 ± 0.79 58.23 7.62 ± 0.89
Blood test
  Total cholesterol (g/L) 13.11 2.07 ± 0.55 13.75 2 ± 0.71 12.9 2.1 ± 0.49
  LDL cholesterol (g/L) 4.41 1.21 ± 0.41 3.72 1.1 ± 0.43 4.63 1.23 ± 0.4
  HDL cholesterol (g/L) 5.09 0.52 ± 0.19 3.96 0.52 ± 0.28 5.46 0.52 ± 0.16
  Blood glucose (g/L) 30.18 1 ± 0.56 31.7 1.07 ± 0.64 29.68 0.97 ± 0.53
  Urea (g/L) 20.84 0.49 ± 0.25 20.74 0.5 ± 0.18 20.88 0.49 ± 0.27
  Creatinine level (mg/L) 33.56 11.06 ± 3.82 37.06 12.5 ± 3.94 32.42 10.53 ± 3.64
Cognitive function
  Folstein test (points) 20.84 19.62 ± 7.46 22.14 22 ± 6.23 20.42 18.78 ± 7.69
  SPMSQ (points) 49.26 6.22 ± 3.57 51.28 7.62 ± 3.54 48.60 5.77 ± 3.47

New Table 3:

We have also corrected gene names that were not italicized in the Discussion.

“It allowed the identification of APOE (p < 0.001) and ACE (p < 0.01) as the first genes associated with human longevity in 1994 using a candidate gene approach in a case–control study including 338 centenarians and 410 adults aged 20–70 years [22]. However, while APOE association was largely replicated in many other studies [2324], ACE association should be considered as a false positive signal due to genotyping issues [32].” was corrected to “It allowed the identification of APOE (p < 0.001) and ACE (p < 0.01) as the first genes associated with human longevity in 1994 using a candidate gene approach in a case–control study including 338 centenarians and 410 adults aged 20–70 years [22]. However, while APOE association was largely replicated in many other studies [2324], ACE association should be considered as a false positive signal due to genotyping issues [32].”

Footnotes

Publisher's Note

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Contributor Information

Alexandre How‑Kit, Email: alexandre.how-kit@fjd-ceph.org.

Jean‑François Deleuze, Email: deleuze@cnrgh.fr.


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