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. 2001 Jan 6;322(7277):46.

Risk of torsades de pointes with non-cardiac drugs

Prolongation of QT interval is probably a class effect of fluoroquinolones

Paul B Iannini 1,2, Sanjay Doddamani 1,2, Eteri Byazrova 1,2, Iulia Curciumaru 1,2, Harvey Kramer 1,2
PMCID: PMC1119313  PMID: 11141163

Editor—Yap and Camm emphasise the risk of torsades de pointes associated with non-cardiac drugs that prolong the QT interval.1 They comment on the fluoroquinolone antimicrobial agents grepafloxacin and sparfloxacin causing QT prolongation but also the apparent lack of this effect with levofloxacin. We recently cared for a patient who developed torsades de pointes while taking levofloxacin, which prompted us to examine retrospectively paired electrocardiograms in other patients to compare QTc intervals before and after they started treatment with this drug.

Twenty three patients who received a standard dose of 500 mg levofloxacin daily had cardiograms that could be compared for QTc prolongation. Prolongation of >30 ms was found in four patients and of >60 ms in two patients. Absolute QT interval prolongation of >500 ms was present in four, one of whom developed torsades de pointes. This patient was also receiving amiodarone, which is known to prolong the QTc interval but not commonly associated with pro-arrhythmia when used alone.

The United States Food and Drug Administration's spontaneous reporting system documents 11 other cases of torsades de pointes in patients receiving levofloxacin, and Samaha reported on an additional patient who also was receiving amiodarone.2 Studies in rabbit Purkinje fibres have shown a concentration-dependent prolongation of the maximal rate of depolarisation at concentrations of ofloxacin up to 100 μmol/l, but the trend did not achieve significance.3 Serum concentrations of 8 μmol/l are achieved by levofloxacin, but the affinity for cardiac tissues is unknown. The Food and Drug Administration has recently requested studies of levofloxacin on cardiac electrophysiology.

Our data indicate that prolongation of the QT interval is probably a class effect of the fluoroquinolones including levofloxacin. Care should be taken when this agent is used with type IA and type III antiarrhythmic agents and in situations such as hypokalaemia and hypomagnesaemia that increase the risk of pro-arrhythmic events.

Footnotes

Competing interests: PBI is a consultant for Bristol-Myers Squibb, Aventis, SmithKline Beecham, Bayer, Roche, Glaxo, TAP, and Abbott pharmaceutical companies. None declared for SD, EB, IC, and HK.

References

  • 1.Yap YG, Camm J. Risk of torsades de pointes with non-cardiac drugs. BMJ. 2000;320:1158–1159. doi: 10.1136/bmj.320.7243.1158. . (29 April.) [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 2.Samaha FF. QTc interval prolongation and polymorphic ventricular tachycardia in association with levofloxacin. Am J Med. 1999;107:528–529. doi: 10.1016/s0002-9343(99)00171-0. [DOI] [PubMed] [Google Scholar]
  • 3.Adamantidis MM, Dumotier BM, Caron JF, Bordet R. Sparfloxacin but not levofloxacin or ofloxacin prolongs cardiac repolarization in rabbit Purkinje fibers. Fundam Clin Pharmacol. 1998;12:70–76. doi: 10.1111/j.1472-8206.1998.tb00926.x. [DOI] [PubMed] [Google Scholar]
BMJ. 2001 Jan 6;322(7277):46.

Grapefruit juice is source of potentially life threatening adverse drug reactions

Phillip Marinucci 1

Editor—Yap and Camm state that “In clinical practice, adverse effects of QT prolonging drugs can be prevented by not exceeding the recommended dose; by restricting the dose in patients with pre-existing heart disease or other risk factors; and by avoiding concomitant administration of drugs that inhibit drug metabolism or excretion, prolong the QT interval, or produce hypokalaemia.”1-1 This leaves out one important point.

Grapefruit juice inhibits the same cytochrome P450 as the imidazoles and macrolides.1-2 As a result, QT prolongation from interactions between these drugs and cisapride or terfenadine (among others) is just as likely as QT prolongation from interactions between grapefruit juice and cisapride or terfenadine. It was my understanding that the regulatory changes that led to terfenadine being removed from the shelves and made a prescription only drug were partly a result of this effect.1-3

The effect was first noted in 1989 when grapefruit juice was used as a masking agent for the taste of felodipine in a small study on the interaction between alcohol and felodipine.1-4 The effect has been widely studied and involves not only cisapride and felodipine (but not non-dihydropyridine calcium channel blockers) but also carbamazepine, caffeine (but not theophylline), ethinyloestradiol, cyclosporin, coumarin, some of the statins, saquinavir, and some benzodiazepines.1-1,1-5

The effect of grapefruit juice is perhaps not as well known or appreciated as it should be and is a source of potentially life threatening adverse drug reactions that are easily avoidable. Doctors and the general public need to be aware of this effect as even seemingly harmless drugs may lead to serious effects.

Footnotes

Competing interests: None declared.

References


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