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editorial
. 2002 May 25;324(7348):1227–1228. doi: 10.1136/bmj.324.7348.1227

Postpartum urinary incontinence

The problem is clear, but there is no simple solution

Linda Brubaker 1,2
PMCID: PMC1123200  PMID: 12028960

Postpartum urinary incontinence is an important and often overlooked form of maternal morbidity. In this issue Chiarelli and Cockburn (p 1241)1 highlight and confirm the work of other investigators who have shown that vaginal delivery induces urinary incontinence, especially the first vaginal birth.2 Many clinical studies have attempted to discover the particular obstetric event that causes the incontinence. The obvious suspects include large babies and “difficult deliveries” marked by lengthy pushing phases with or without instrumentation. No clear single event has been found to be responsible, suggesting that postpartum urinary incontinence arises from a multifactorial physiological insult. The consequences of this pathophysiology are not limited to urinary incontinence. Pelvic organ prolapse (cystocele, rectocele, and uterine prolapse) and anal incontinence are also troublesome sequelae of vaginal delivery. These prevalent pelvic problems receive even less than the scant attention paid to postpartum urinary incontinence.3,4

While the problem is clear, there is no simple solution. Prevention is rarely discussed among caregivers of urinary incontinence possibly because at this time the price of prevention is major surgery. Several studies suggest even this protection may fade with repeated abdominal deliveries.5 It is understandable that this method of prevention has not been met with widespread support. Avoidance or modification of specific obstetric techniques has not been shown to prevent postpartum urinary incontinence. In this vacuum of scientific uncertainty, emotion rapidly fills the void. Patients and physicians alike respond to the scientific uncertainty with preferences based on their personal convictions.6

If we cannot prevent the damage that causes postpartum urinary incontinence, it is reasonable to attempt to mitigate the damage. Chiarelli and Cockburn conducted a randomised trial to see if instructions to patients and postpartum pelvic rehabilitation would be beneficial. They provided new mothers with a comprehensive bladder programme, including enhanced information about healthy bladder habits and teaching with reinforcement regarding muscle training. Although the authors report a slight effect, the reader is struck by the high rates of incontinence even with such conscientious rehabilitation efforts: only 7% of incontinent new mothers reduced symptoms, leaving most symptomatic women untreated. Even these excellent efforts were grossly insufficient for the many new mothers who develop postpartum urinary incontinence, and these young women are likely to continue to experience the indignity of urinary incontinence for many decades to come.7

We are left pondering the appropriate individual balance between vaginal birth and pelvic floor health. Large, longitudinal studies with well characterised populations and carefully described outcome measures will be essential to gather information for counselling patients. Recommendations about routes of delivery or the conduct of labour must include maternal pelvic floor morbidity, using appropriate windows of postpartum observation. Surgical trials would not be published if the authors reported surgical morbidity only during the procedure. Obstetric providers must look broadly at pelvic morbidity in order to optimise the care of maternity patients.

Clearly the risk of postpartum urinary incontinence exists, and abdominal delivery without labour markedly reduces the risk. Ongoing research will give us additional information about individual patients who are particularly susceptible to damage, possibly because of their constitutional make up or their particular obstetric situation. Meanwhile, how shall we counsel patients, especially those who particularly abhor the risks of pelvic floor damage?

Each woman should have sufficient information to determine which set of risks she prefers for herself and her baby. In the common situation where there is no additional risk to a baby, obstetric management should focus on reducing maternal morbidity including postpartum urinary incontinence. New mothers are likely to benefit from routine symptom screening and early discussion of healthy bladder habits and proper muscle techniques as part of their postpartum care. Obstetric care should include assessment of the maternal outcome of that birth, including the complete range of pelvic floor injuries known to be associated with childbirth.

Chiarelli and Cockburn are to be commended for bringing high quality science to the neglected problem of postpartum urinary incontinence. This problem cries for attention and a scientific approach to prevention or early intervention. Rather than confining our intellectual discourse to a dichotomous discussion regarding route of delivery, creative approaches are needed to develop a balance that optimises both maternal and child health. Some say that the mark of a civilised nation is the care that the new mother and her child receive. With one of three new mothers becoming incontinent of urine, how would we be judged?

Papers p 1241

References

  • 1.Chiarelli P, Cockburn J. Promoting urinary continence in women after delivery: randomised controlled trial. BMJ. 2002;324:1241–1244. doi: 10.1136/bmj.324.7348.1241. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 2.Farrell SA, Allen VM, Baskett TF. Parturition and urinary incontinence in primiparas. J Obstet Gynecol. 2001;97:350–356. doi: 10.1016/s0029-7844(00)01164-9. [DOI] [PubMed] [Google Scholar]
  • 3.Chaliha C, Sultan AH, Bland JM, Monga AK, Stanton SL. Anal function: effect of pregnancy and delivery. Am J Obstet Gynecol. 2001;185:427–432. doi: 10.1067/mob.2001.115997. [DOI] [PubMed] [Google Scholar]
  • 4.Donnelly V, Fynes M, Campbell D, Johnson H, O'Connell PR, O'Herlihy C. Obstetric events leading to anal sphincter damage. Obstet Gynecol. 1998;92:955–961. doi: 10.1016/s0029-7844(98)00255-5. [DOI] [PubMed] [Google Scholar]
  • 5.MacLennan AH, Taylor AW, Wilson DH, Wilson D. The prevalence of pelvic floor disorders and their relationship to gender, age, parity and mode of delivery. Br J Obstet Gynecol. 2000;107:1460–1470. doi: 10.1111/j.1471-0528.2000.tb11669.x. [DOI] [PubMed] [Google Scholar]
  • 6.Al-Mufti R, McCatrhy A, Fist NM. Survey of obstetrician's personal preference and discretionary practice. Eur J Obstet Gynecol Reprod Biol. 1997;73:1–4. doi: 10.1016/s0301-2115(96)02692-9. [DOI] [PubMed] [Google Scholar]
  • 7.Viktrup L. The risk of lower urinary tract symptoms five years after the first delivery. Neurourol Urodyn. 2002;21:2–29. doi: 10.1002/nau.2198. [DOI] [PubMed] [Google Scholar]

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