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Cancer Cell International logoLink to Cancer Cell International
. 2024 Jul 23;24:260. doi: 10.1186/s12935-024-03399-x

Correction: MALAT1/ mir-1-3p mediated BRF2 expression promotes HCC progression via inhibiting the LKB1/AMPK signaling pathway

Guang-Zhen Li 1,2,#, Guang-Xiao Meng 2,3,#, Guo-Qiang Pan 2,3, Xiao Zhang 2,3, Lun-Jie Yan 2,3, Rui-Zhe Li 2,3, Zi-Niu Ding 2, Si-Yu Tan 2, Dong-Xu Wang 2, Bao-wen Tian 2, Yu-Chuan Yan 2,3, Zhao-Ru Dong 2, Jian-Guo Hong 2,, Tao Li 2,
PMCID: PMC11264463  PMID: 39044182

Correction to: Cancer Cell International (2023) 23:188

10.1186/s12935-023-03034-1.

In this article [1], the wrong figure appeared as Fig. 7, the figure should have appeared as shown below.

Fig. 7.

Fig. 7

Overexpression of BRF2 abrogated MALAT1 knockdown. (a, b) The transfection efficiency of OE-BRF2 in Huh7 and Hep3B cells was detected by qRT-PCR. (c, d) After silencing MALAT1 and overexpression BRF2, cell viability of Huh7 and Hep3B cells was detected by CCK-8 assay on days 0, 1, 2, 3, 4 and 5. (e, f) Transwell assay was used to detect migration and invasion of HCC cells after MALAT1 down-regulation and overexpression of BRF2. (g) The apoptosis rate of HCC cells after MALAT1 down-regulation and overexpression of BRF2 was detected by flow cytometry. (h) Western blot analysis was performed to detect the expression levels of apoptosis-related proteins and LKB1/AMPK in HCC cells after MALAT1 down-regulation and overexpression of BRF2. **P < 0.01, ***P < 0.001

Footnotes

The online version of the original article can be found at 10.1186/s12935-023-03034-1.

Publisher’s Note

Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.

Guang-Zhen Li and Guang-Xiao Meng contributed equally to this work.

Contributor Information

Jian-Guo Hong, Email: 200862000175@sdu.edu.cn.

Tao Li, Email: litao7706@163.com.

References

  • 1.Li GZ, Meng GX, Pan GQ, Zhang X, Yan LJ, Li RZ, Ding ZN, Tan SY, Wang DX, Tian BW, Yan YC. MALAT1/mir-1-3p mediated BRF2 expression promotes HCC progression via inhibiting the LKB1/AMPK signaling pathway. Cancer Cell Int. 2023;23(1):188. [DOI] [PMC free article] [PubMed]

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