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. 2024 Jul 30;25:745. doi: 10.1186/s12864-024-10660-0

Correction: HiFi long-read amplicon sequencing for full-spectrum variants of human mtDNA

Yan Lin 1,#, Jiayin Wang 1,#, Ran Xu 2,#, Zhe Xu 3, Yifan Wang 2, Shirang Pan 2, Yan Zhang 2, Qing Tao 2, Yuying Zhao 1, Chuanzhu Yan 1,4,5, Zhenhua Cao 2,✉,#, Kunqian Ji 1,6,#
PMCID: PMC11289926  PMID: 39080517

Correction: BMC Genomics 25, 538 (2024)

10.1186/s12864-024-10433-9

In this article, an older version of Figure 5 was submitted for publication. The correct and incorrect version of Fig. 5 is given below. The figure caption remains unchanged.

Fig. 5.

Fig. 5

SV circle diagram of myositis patient and muscle histological and histochemical pathological images of P20. (A). P1-P6, and P20 all were detected with SV and SV with a ratio > 4% circle plots. (B). Muscle histology and histochemistry suggested mitochondrion dysfunctions in P20. In the first-line pictures, HE, MGT, COX, and SDH/COX double staining showed the features of mitochondrial dysfunctions. In the second line, the infiltrates of CD3+ and CD68+ cells, along with the expressions of MHC-1 and MAC, were consistent with pathological changes in inflammatory myopathy. HE: hematoxylin and eosin; MGT: modified Gomori trichrome; COX: cytochrome C oxidase; SDH: succinate dehydrogenase; S/C: SDH/COX double histochemistry; MHC-I: anti-major histocompatibility complex class I; MxA: myxovirus resistant protein A

Correct

Incorrect

graphic file with name 12864_2024_10660_Fig2_HTML.jpg

The original article has been corrected.

The authors would like to apologize for any inconvenience caused to the readers.

Footnotes

The online version of the original article can be found at 10.1186/s12864-024-10433-9.

Publisher’s Note

Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.

Yan Lin, Jiayin Wang and Ran Xu contributed equally to this work.

Zhenhua Cao and Kunqian Ji contributed equally to this work.


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