Corrigendum to “Platelets and Hemostatic Proteins are Co-Localized with Chronic Neuroinflammation Surrounding Implanted Intracortical Microelectrodes” [Acta Biomaterialia. Volume 166, August 2023, Pages 278–290]

The authors regret the oversight in which select citations contained errors that were overlooked during the review and editorial process. The authors have made the appropriate corrections. It is important to note that these corrections do not impact the data and conclusions presented in the study. The integrity and validity of the findings remain unchanged.

The manuscript version available on PubMed remains unchanged. The below correction applies to the version available on ScienceDirect.

The impacted sentences with amended in-line citations as well as the entire corrected bibliography are reproduced below.

The authors apologise for any inconvenience caused.

Although platelets’ primary function is to prevent vascular leaks and promote wound healing, under inflammatory conditions activated platelets have been suggested to promote leukocytic migration through modulating endothelial junctions [46–50,55,56].

Furthermore, fibrinogen-induced neuroinflammation has been linked to neurologic disease models such as Multiple Sclerosis, Alzheimer’s Disease, and ischemic stroke [89,107,108].

Sustained platelet presence may be influenced by a prolonged dysfunctional BBB and inflammatory chemokines released by activated endothelial and immune cells [55,71,112].