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. 2024 Sep 30;13(19):1630. doi: 10.3390/cells13191630

Correction: Pittala et al. The VDAC1-based R-Tf-D-LP4 Peptide as a Potential Treatment for Diabetes Mellitus. Cells 2020, 9, 481

Srinivas Pittala 1, Idan Levy 1, Soumasree De 1, Swaroop Kumar Pandey 1, Nataly Melnikov 1, Tehila Hyman 1, Varda Shoshan-Barmatz 1,*
PMCID: PMC11475749  PMID: 39404435

Error in Figure

In the original publication [1], the image in Figure 1D is overlapping with Figure 1E [2]. The images have been inadvertently overleaped with previously published work. The corrected Figure 1D, R-Tf-D-LP4 has now been replaced with a different image, shown below.

Figure 1.

Figure 1

R-Tf-D-LP4 peptide-mediated inhibition of steatotic and non-alcoholic steatohepatitis (NASH) liver pathology in a STZ/HFD-32 mouse model. (A). Schematic presentation of the course of steatosis and NASH induced by a STZ/HFD-32 diet and the effect of R-Tf-D-LP4 peptide treatment. (B–D). Liver from mice fed with chow (normal diet), HFD-32, or HFD-32 and treated with the R-Tf-D-LP4 peptide (14 mg/kg) by i.v. injection every two days from Week 6 to 8 for steatosis and from Week 8 to 10 for NASH, as described in the Methods section. Mice were then sacrificed, livers were removed, photographed (B), and weighed (C) Results are means ± SEM (n = 10), (p **** ≤ 0.0001). Representative liver sections were stained with Oil Red O (D). Blood glucose level of mice was measured. Results are means ± SEM (n = 5–10; ** p ≤ 0.01, p **** ≤ 0.0001) (E).

The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

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References

  • 1.Pittala S., Levy I., De S., Kumar Pandey S., Melnikov N., Hyman T., Shoshan-Barmatz V. The VDAC1-based R-Tf-D-LP4 Peptide as a Potential Treatment for Diabetes Mellitus. Cells. 2020;9:481. doi: 10.3390/cells9020481. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 2.Pittala S., Krelin Y., Kuperman Y., Shoshan-Barmatz V. A Mitochondrial VDAC1-Based Peptide Greatly Suppresses Steatosis and NASH-Associated Pathologies in a Mouse Model. Mol. Ther. 2019;27:1848–1862. doi: 10.1016/j.ymthe.2019.06.017. [DOI] [PMC free article] [PubMed] [Google Scholar]

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