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. 1982 May 15;204(2):617–619. doi: 10.1042/bj2040617

Location of the gene for the low-affinity tryptophan-specific permease of Escherichia coli.

R M Edwards, M D Yudkin
PMCID: PMC1158393  PMID: 7052072

Abstract

L-Tryptophan uptake was assayed under conditions in which the aroT gene had been inactivated by deletion and the product of the aroP permease was competitively inhibition. A mutant carrying a deletion from bgl through tnaA showed negligible L-tryptophan uptake, in contrast to a strain possessing an intact tna region or to strains carrying point mutations in tna. The ability to take up L-tryptophan was not restored by lysogenizing the tna-deleted strain with lambda tna+.

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Selected References

These references are in PubMed. This may not be the complete list of references from this article.

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