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. 1999 Nov 15;18(22):6339–6348. doi: 10.1093/emboj/18.22.6339

Loss of receptor regulation by a phospholipase D1 mutant unresponsive to protein kinase C.

Y Zhang 1, Y M Altshuller 1, S M Hammond 1, F Hayes 1, A J Morris 1, M A Frohman 1
PMCID: PMC1171697  PMID: 10562546

Abstract

Activation of phosphatidylcholine-specific phospholipase D (PLD) constitutes an important part of the cellular response to agonist signaling. PLD1 is stimulated in vitro in a direct and synergistic manner by protein kinase C (PKC), ADP-ribosylation factor (ARF) and Rho family members. However, the direct and specific role of each of these effectors in agonist-stimulated PLD activation is poorly understood. We have used transposon mutagenesis to generate a library of PLD1 alleles containing random pentapeptide insertions. Forty-five alleles were characterized to identify functionally important regions. Use of an allele unresponsive to PKC, but otherwise seemingly normal, to examine coupling of PLD1 to a subset of G-protein-coupled receptors demonstrates for the first time direct stimulation of PLD1 in vivo by PKC and reveals that this direct stimulation is unexpectedly critical for PLD1 activation.

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