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. 2025 Jan 25;45:16. doi: 10.1007/s10571-025-01530-8

Correction: Docosahexaenoic Acid Alleviates Oxidative Stress-Based Apoptosis Via Improving Mitochondrial Dynamics in Early Brain Injury After Subarachnoid Hemorrhage

Tongyu Zhang 1,#, Pei Wu 1,#, John H Zhang 2, Yuchen Li 1, Shancai Xu 1, Chunlei Wang 1, Ligang Wang 1, Guang Zhang 1, Jiaxing Dai 1, Shiyi Zhu 1, Yao Liu 1, Binbing Liu 1, Cesar Reis 2, Huaizhang Shi 1,
PMCID: PMC11762597  PMID: 39862294

Correction to: Cellular and Molecular Neurobiology (2018) 38:1413–1423 10.1007/s10571-018-0608-3

The original version of this article unfortunately contained an error in figure.

In Fig. 6, the error is in the presentation of the β-actin protein band as the authors used incorrect image in the article.

The authors also assure that this error does not impact the experimental results or the conclusions of their study.

Incorrect version of Fig. 6graphic file with name 10571_2025_1530_Figa_HTML.jpg

Correct version of Fig. 6

Fig. 6.

Fig. 6

DHA improved the balance of mitochondrial fission and fusion after SAH in vitro and in vivo. Representative western blot images showing the effect of DHA on the expressions of Drp1, p-Drp1 (Ser616), L-OPA1, and S-OPA1. a in vitro and b in vivo. Quantitative analysis of above proteins among all groups showed that DHA treatment decreased the expression of Drp1 and p-Drp1 (Ser616) and increased the expression of L-OPA1 and S-OPA1 c in vitro and d in vivo. n = 6 for each group. Bars represent the mean ± SD. #p < 0.05 versus Control or Sham group; *p < 0.05 versus OxyHb or SAH + vehicle group. Vehicle, 1% dimethylsulfoxide and sterile saline

The original article has been corrected.

Footnotes

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Tongyu Zhang and Pei Wu contributed equally to this work.


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