Correction to: Cell Division (2024) 19:17
10.1186/s13008-024-00122-0
In this article [1], the author would like to correct the errors in Figs. 3, 7 and 8 and Supplement Figs. 1, 3 and 4. For completeness and transparency, the old incorrect and correct versions are displayed below.
Fig. 3.
LncRNA NUTM2A-AS1 negatively regulates miR-376a-3p in U251 cell line. A–C qRT-PCR was performed to analyze the expression of lncRNA NUTM2A-AS1 and miR-376a-3p in U251 cells. D MTT assay was conducted to assess the cell viability of U251 cells. E, F Flow cytometry was used to quantify the apoptosis of U251 cells. G Western blot assay was conducted to analyze the protein expression of Bax and Bcl-2. H qRT-PCR was conducted to analyze the mRNA expression of Bax. I qRT-PCR was conducted to analyze the mRNA expression of Bcl-2. **p < 0.01 vs. control-siRNA; ##p < 0.01 vs. inhibitor control; &&p < 0.01 vs. NUTM2A-AS1-siRNA + inhibitor control
Fig. 7.
MiR-376a-3p affects proliferation and apoptosis of U251 cells through the downregulation of YAP1. A MTT assay was conducted to assess the cell viability of U251 cells. B, C Flow cytometry was used to quantify the apoptosis of U251 cells. D Western blot assay was conducted to analyse the protein expression of Bax and Bcl-2. E qRT-PCR was conducted to analyze the mRNA expression of Bax. F qRT-PCR was conducted to analyse the mRNA expression of Bcl-2. **p < 0.01 vs. mimic control; ##p < 0.01 vs. miR-376a-3p mimic + control-plasmid
Fig. 8.
YAP1 enhances proliferation and reduces apoptosis of U251 cells. A MTT assay was conducted to assess the cell proliferation of U251 cells. B, C Flow cytometry was used to quantify the apoptosis of U251 cells. D Western blot assay was conducted to analyze the protein expression of Bax and Bcl-2. E qRT-PCR was conducted to analyze the mRNA expression of Bax. F qRT-PCR was conducted to analyze the mRNA expression of Bcl-2. **p < 0.01 vs. control-plasmid group
Incorrect Figures:
Fig. 3.
LncRNA NUTM2A-AS1 negatively regulates miR-376a-3p in U251 cell line. A–C qRT-PCR was performed to analyze the expression of lncRNA NUTM2A-AS1 and miR-376a-3p in U251 cells. D MTT assay was conducted to assess the cell viability of U251 cells. E, F Flow cytometry was used to quantify the apoptosis of U251 cells. G Western blot assay was conducted to analyze the protein expression of Bax and Bcl-2. H qRT-PCR was conducted to analyze the mRNA expression of Bax. I qRT-PCR was conducted to analyze the mRNA expression of Bcl-2. **p < 0.01 vs. control-siRNA; ##p < 0.01 vs. inhibitor control; &&p < 0.01 vs. NUTM2A-AS1-siRNA + inhibitor control
Fig. 7.
MiR-376a-3p affects proliferation and apoptosis of U251 cells through the downregulation of YAP1. A MTT assay was conducted to assess the cell viability of U251 cells. B, C Flow cytometry was used to quantify the apoptosis of U251 cells. D Western blot assay was conducted to analyse the protein expression of Bax and Bcl-2. E qRT-PCR was conducted to analyze the mRNA expression of Bax. F qRT-PCR was conducted to analyse the mRNA expression of Bcl-2. **p < 0.01 vs. mimic control; ##p < 0.01 vs. miR-376a-3p mimic + control-plasmid
Fig. 8.
YAP1 enhances proliferation and reduces apoptosis of U251 cells. A MTT assay was conducted to assess the cell proliferation of U251 cells. B, C Flow cytometry was used to quantify the apoptosis of U251 cells. D Western blot assay was conducted to analyze the protein expression of Bax and Bcl-2. E qRT-PCR was conducted to analyze the mRNA expression of Bax. F qRT-PCR was conducted to analyze the mRNA expression of Bcl-2. **p < 0.01 vs. control-plasmid group
Correct Figures:
Electronic supplementary material
Below is the link to the electronic supplementary material
Footnotes
Publisher’s note
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Yuecheng Zeng and Zhenyu Yang are equal contributors.
Contributor Information
Yang Yang, Email: yy195028@163.com.
Peng Wang, Email: 24954614@qq.com.
Reference
- 1.Zeng Y, Yang Z, Yang Y, Wang P. LncRNA NUTM2A-AS1 Silencing inhibits glioma via miR-376a-3p/YAP1 axis. Cell Div. 2024;19:17. [DOI] [PMC free article] [PubMed] [Google Scholar]
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