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. Author manuscript; available in PMC: 2025 Sep 12.
Published in final edited form as: DNA Repair (Amst). 2025 Aug 5;153:103878. doi: 10.1016/j.dnarep.2025.103878

Corrigendum to “Unveiling cGAS mechanisms: Insights into DNA damage and immune sensing in cancer” [DNA Repair 152 (2025) 103868]

Min-Guk Cho a,*, Gaorav P Gupta a,b,c,d,e
PMCID: PMC12424580  NIHMSID: NIHMS2106256  PMID: 40753005

The authors regret that the originally published Figs. 1, 2 and 4 were of suboptimal quality and would like to provide a higher-resolution version to improve clarity.

Fig. 1.

Fig. 1

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Fig. 2.

Fig. 2

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Fig. 4.

Fig. 4

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The improved figures are provided below:

In addition, the authors would like to revise the highlights section as follows:

The authors would like to apologise for any inconvenience caused.

Revised Highlights.

DNA damage promotes the release of dsDNA from nuclear, mitochondrial, and micronuclear sources, activating the cGAS-STING pathway.

sensing, potentially requiring PTMs and nuclear export.

Intracellular trafficking may control cGAS-STING signaling to balance immune activation and tolerance.

STING agonists are proposed to enhance tumor immunity, but challenges include delivery, toxicity, and context-dependent immune suppression.

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