In this article, four references (references [6],[60],[92],[117]) required revisions due to issues of comprehensiveness, retraction, methodological rigor, and contextual appropriateness. These corrections ensure the accuracy and reliability of the cited sources.
Original versions of references [6],[60],[92],[117]:
[6] H. Valadi, K. Ekström, A. Bossios, M. Sjöstrand, J.J. Lee, J.O. Lötvall, Exosome-mediated transfer of mRNAs and microRNAs is a novel mechanism of genetic exchange between cells, Nat. Cell Biol., 9 (2007), pp. 654–659.
[60] T. Deng, H. Zhang, H. Yang, H. Wang, M. Bai, W. Sun et al., Exosome miR-155 derived from gastric carcinoma promotes angiogenesis by targeting the c-MYB/VEGF Axis of endothelial cells, Mol. Ther. Nucleic Acids 19 (2020) 1449–1459.
[92] A. Casado-Díaz, J.M. Quesada-Gomez, G. Dorado, Extracellular vesicles derived from mesenchymal stem cells (MSC) in regenerative medicine: applications in skin wound healing, Front. Bioeng. Biotechnol. 8 (2020) 146.
[117] Y.G. Chen, R. Chen, S. Ahmad, R. Verma, S.P. Kasturi, L. Amaya et al., N6-Methyladenosine modification controls circular RNA immunity, Mol. Cell. 76 (2019) 96–109.e9.
References [6],[92], and [117] were replaced with new references, respectively.
The retracted reference [60] was fully removed from the citation list.
The correct versions of the References [6],[92], and [117] should be as below.
[6] W. Li, X. Wang, C. Li, T. Chen, Q. Yang, Exosomal Non-Coding RNAs: Emerging Roles in Bilateral Communication Between Cancer Cells and Macrophages, Mol. Ther. 30 (2022), pp. 1036–1053.
[92] M. El-Tanani, H. Nsairat, I.I. Matalka et al., Impact of Exosome Therapy on Pancreatic Cancer and Its Progression, Med. Oncol. 40 (2023), pp. 225.
[117] P. Ding, H. Wu, J. Wu et al., N6-Methyladenosine Modified CircPAK2 Promotes Lymph Node Metastasis via Targeting IGF2BPs/VEGFA Signaling in Gastric Cancer, Oncogene 43 (2024), pp. 2548–2563.
The authors apologize for the errors.
Declaration of competing interest
The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.