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. 2025 Sep 17;13:1685169. doi: 10.3389/fchem.2025.1685169

Correction: Investigation of the mechanism of Xiaoyin Jiedu Yin in the treatment of psoriasis based on bioinformatics, machine learning

Ru-Nan Fang 1, Yang Zhou 2, Yang Shen 1, Yuan Sun 1, Jian-Hong Li 1,*
PMCID: PMC12487649  PMID: 41040846

There was a mistake in Figure 6B as published. The β-Tubulin bands were indentified as target protein, and the AKR1B10 bands were identified as internal control. The β-Tubulin bands should serve as the internal control, while the AKR1B10 bands represent the target protein. The corrected figure appears below.

FIGURE 6.

Panel A shows a bar graph of HaCaT cell proliferation rates with varying concentrations of XYJDY serum, indicating significant differences among groups. Panel B displays protein expression bands for AKR1B10 and β-Tubulin across different conditions. Panel C presents a bar graph of relative protein expression adjusted for β-Tubulin, comparing control, psoriasis, and XYJDY with psoriasis groups. Panel D shows a bar graph of relative AKR1B10 mRNA levels, highlighting significant differences among conditions. Statistical significance is noted with asterisks.

(A) CCK-8 Detection of cell proliferation rate in a psoriasis-like inflammation model of HaCaT cells after 24 24 h of intervention with XYJDY drug-containing serum; (B) Western blot analysis of AKR1B10 expression in HaCaT cells induced by IL-17A in different treatments; (C) Western blot detection of AKR1B10 expression in each group (n = 3/group); (D) RT-qPCR analysis to detect the expression of AKR1B10 mRNAs in each group (n = 3/group); **P < 0.01, ***P < 0.001, ****P < 0.0001.

The original article has been updated

Footnotes

Edited and reviewed by: Ahmed A. Al-Karmalawy, University of Mashreq, Iraq

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