Correction to: Cell Mol Neurobiol (2015) 35:891–898
10.1007/s10571-015-0184-8
The original version of this article unfortunately contained error in Fig. 1.
In Fig. 1A, flow cytometry images actually belongs to authors’ another manuscript as the data from two projects conducted around same time has resulted in this confusion.
In addition, in Fig. 1E, five flow cytometry images are displayed while only four experimental groups are included in the figure. The last image is a duplicate image of the same “Sev” group, and should be removed for clarity.
Incorrect version of Fig. 1
Fig. 1.
Propofol attenuates Sev-induced apoptosis and ROS production. a, b Apoptosis attenuation induced by propofol in Sevstimulated H4 cells. Apoptosis induced by Sev or attenuated by propofol was tested by FACS analysis after staining with Annexin V-FITC. Apoptotic cells were fewer in propofol treated cells than that in Sev-stimulated H4 cells (a) and the index of apoptosis was showed in (b). c, d The expression levels of apoptosis-related protein. Levels of cleaved caspase-3, Bcl-2, and Bax after Sev and/or propofol treatment in H4 cells were tested and compared by Western blot analysis (c), and the quantitative analysis of gray intensity was calculated and showed in (d). e, f Propofol inhibited ROS production induced by Sev in H4 cells. ROS content was measured with 2,7'- dihydrofluorescein diacetate (DCFH-DA) after treatment with Sev and/or propofol in H4 cells. Oxidized dihydrofluorescein (DCF) levels were analyzed by FACS. The peak shift to the right indicates increased levels of ROS content meanwhile to the left indicates decreased levels (e). The comparative analysis of ROS levels was calculated and showed in (f). **P < 0.01 versus Sev group; ##P < 0.01 versus control group. Sev sevoflurane, Pro propofol
Correct version of Fig. 1
Fig. 1.
Propofol attenuates Sev-induced apoptosis and ROS production. a, b Apoptosis attenuation induced by propofol in Sevstimulated H4 cells. Apoptosis induced by Sev or attenuated by propofol was tested by FACS analysis after staining with Annexin V-FITC. Apoptotic cells were fewer in propofol treated cells than that in Sev-stimulated H4 cells (a) and the index of apoptosis was showed in (b). c, d The expression levels of apoptosis-related protein. Levels of cleaved caspase-3, Bcl-2, and Bax after Sev and/or propofol treatment in H4 cells were tested and compared by Western blot analysis (c), and the quantitative analysis of gray intensity was calculated and showed in (d). e, f Propofol inhibited ROS production induced by Sev in H4 cells. ROS content was measured with 2,7'- dihydrofluorescein diacetate (DCFH-DA) after treatment with Sev and/or propofol in H4 cells. Oxidized dihydrofluorescein (DCF) levels were analyzed by FACS. The peak shift to the right indicates increased levels of ROS content meanwhile to the left indicates decreased levels (e). The comparative analysis of ROS levels was calculated and showed in (f). **P < 0.01 versus Sev group; ##P < 0.01 versus control group. Sev sevoflurane, Pro propofol
The original article has been corrected.
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