Correction to: Molecular Biology Reports (2024) 51: 995
10.1007/s11033-024-09928-1
In this article, Figs. 2, 3, 5D, 6D and 7D appeared incorrectly and have now been corrected in the original publication. For completeness and transparency, the correct and old incorrect versions are displayed below.
Fig. 2.
CAFs promote PSN1 cells proliferation, invasion and migration. A CCK8 assay was performed in PSN1 cells. B Colony formation assay was performed in PSN1 cells. C The transwell assay was used to assess PSN1 cells’ capacity for invasion. D Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by one-way ANOVA, and the Sidak test was used as post hoc. *p < 0.05; **p < 0.01; ***p < 0.001
Fig. 3.
CAFs promote PSN1 cells proliferation, invasion, and migration by secreting exosomes. A CCK8 assay was performed in PSN1 cells. B Colony formation assay was performed in PSN1 cells. C Transwell assay was performed to evaluate the invasive ability of PSN1 cells. D Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by one-way ANOVA, and the Sidak test was used as post hoc. *p < 0.05; **p < 0.01; ***p < 0.001
Fig. 5.
CAFs exosomes promote PSN1 proliferation, invasion and migration via Leptin. A CCK8 assay was performed in PSN1 cells. B Western blot analysis of Leptin in PSN1. C Colony formation assay was performed in PSN1 cells. D The Transwell assay was used to assess PSN1 cells’ capacity for invasion. E Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by Student’s t-test. *p < 0.05; **p < 0.01; ***p < 0.001
Fig. 6.
miR-224-3p mitigates the inhibitory effect of CAFs knockdown of Leptin on PSN1 cells development. A CCK8 assay was performed in PSN1 cells. B RT-qPCR analysis of miR-224-3p. C Colony formation assay was performed in PSN1 cells. D The Transwell assay was used to assess PSN1 cells’ capacity for invasion. E Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by Student’s t-test. *p < 0.05; **p < 0.01; ***p < 0.001
Fig. 7.
CAFs exosomal Leptin promotes PSN1 cells by inhibiting ABL2 expression through upregulation of miR-224-3p. A PSN1 cells were used for the CCK8 test. B Western blot examination of PSN1’s ABL2. C PSN1 cells were used for the colony formation experiment. D The Transwell assay was used to assess PSN1 cells’ capacity for invasion. E Assays for wound healing were conducted to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by one-way ANOVA, and the Sidak test was used as post hoc. *p < 0.05; **p < 0.01; ***p < 0.001
Incorrect Fig. 2.
Correct Fig. 2.
Fig. 2.
CAFs promote PSN1 cells proliferation, invasion and migration. A CCK8 assay was performed in PSN1 cells. B Colony formation assay was performed in PSN1 cells. C The transwell assay was used to assess PSN1 cells’ capacity for invasion. D Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by one-way ANOVA, and the Sidak test was used as post hoc. *p < 0.05; **p < 0.01; ***p < 0.001
Incorrect Fig. 3.
Correct Fig. 3.
Fig. 3.
CAFs promote PSN1 cells proliferation, invasion, and migration by secreting exosomes. A CCK8 assay was performed in PSN1 cells. B Colony formation assay was performed in PSN1 cells. C Transwell assay was performed to evaluate the invasive ability of PSN1 cells. D Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by one-way ANOVA, and the Sidak test was used as post hoc. *p < 0.05; **p < 0.01; ***p < 0.001
Incorrect Fig. 5.
Correct Fig. 5.
Fig. 5.
CAFs exosomes promote PSN1 proliferation, invasion and migration via Leptin. A CCK8 assay was performed in PSN1 cells. B Western blot analysis of Leptin in PSN1. C Colony formation assay was performed in PSN1 cells. D The Transwell assay was used to assess PSN1 cells’ capacity for invasion. E Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by Student’s t-test. *p < 0.05; **p < 0.01; ***p < 0.001.
Incorrect Fig. 6.
Correct Fig. 6.
Fig. 6.
miR-224-3p mitigates the inhibitory effect of CAFs knockdown of Leptin on PSN1 cells development. A CCK8 assay was performed in PSN1 cells. B RT-qPCR analysis of miR-224-3p. C Colony formation assay was performed in PSN1 cells. D The Transwell assay was used to assess PSN1 cells’ capacity for invasion. E Assays for wound healing were used to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by Student’s t-test. *p < 0.05; **p < 0.01; ***p < 0.001.
Incorrect Fig. 7.
Correct Fig. 7.
Fig. 7.
CAFs exosomal Leptin promotes PSN1 cells by inhibiting ABL2 expression through upregulation of miR-224-3p. A PSN1 cells were used for the CCK8 test. B Western blot examination of PSN1’s ABL2. C PSN1 cells were used for the colony formation experiment. D The Transwell assay was used to assess PSN1 cells’ capacity for invasion. E Assays for wound healing were conducted to evaluate PSN1 cells’ capacity for migration. The data is shown as mean ± SD. Analyses were performed by one-way ANOVA, and the Sidak test was used as post hoc. *p < 0.05; **p < 0.01; ***p < 0.001.
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