PDIA3 orchestrates effector T cell program by serving as a chaperone to facilitate the non-canonical nuclear import of STAT1 and PKM2

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(Molecular Therapy 32, 2778–2797; August 2024)

Inappropriate manipulation occurred during the assembly of images in this study. Specifically, (1) in Figure 1L, the representative flow cytometry plot for the experimental group was mistakenly replaced with that of the control group due to an unsuccessful copy-and-paste operation, and (2) in Figure 3C, the gating boundaries in the flow cytometry plots were misaligned during figure arrangement. These errors have been rectified using the original data, and the corrected figures are presented below. The results and conclusions of the paper remain unaffected by these corrections. The authors sincerely apologize for these inadvertent errors in figure preparation.

Figure 1. PDIA3 is upregulated in arthritogenic CD4 T cells and positively correlates with RA severity (original)

Figure 1. PDIA3 is upregulated in arthritogenic CD4 T cells and positively correlates with RA severity (corrected)

Figure 3. Pdia3 deficiency intrinsically impairs Th1 and Th17 program (original)

Figure 3. Pdia3 deficiency intrinsically impairs Th1 and Th17 program (corrected)