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Oxidative Medicine and Cellular Longevity logoLink to Oxidative Medicine and Cellular Longevity
. 2026 Mar 25;2026:9756127. doi: 10.1155/omcl/9756127

Correction to “Rh‐CXCL‐12 Attenuates Neuronal Pyroptosis after Subarachnoid Hemorrhage in Rats via Regulating the CXCR4/NLRP1 Pathway”

PMCID: PMC13140413  PMID: 41882919

R. Gu, L. Wang, H. Zhou, X. Wang, C. Lenahan, H. Qu, Y. Liu, S. Li, C. Wei, L. Han, X. Hu, G. Zuo, “Rh‐CXCL‐12 Attenuates Neuronal Pyroptosis after Subarachnoid Hemorrhage in Rats via Regulating the CXCR4/NLRP1 Pathway,” Oxidative Medicine and Cellular Longevity, 2021, 6966394, https://doi.org/10.1155/2021/6966394.

In the article, there are errors in Figure 6, introduced by the authors during the figure assembly and highlighted on PubPeer [1]. The correct Figure 6 is shown below:

Figure 6.

Figure 6

Intranasal administration of rh‐CXCL‐12 attenuated CC‐1‐positive neurons after SAH. (a) Representative immunofluorescence staining of CC‐1‐positive neurons in the ipsilateral basal cortex at 24 h after SAH. Green indicated CC1‐positive staining, red indicated neurons, and blue indicated DAPI‐positive nuclear staining. A small black square within the coronal section of the brain indicated the location of where the immunofluorescence staining images were taken. (b) Quantitative analysis of CC‐1‐positive neurons. n = 4 per group. Vehicle: sterile distilled water. Data were represented as mean ± SD.  ∗ P < 0.05 vs. sham group; @ P < 0.05 vs. SAH + vehicle group.

We apologize for these errors.

References


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