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. 2026 May 20;17:1848575. doi: 10.3389/fgene.2026.1848575

Correction: SPDL1 overexpression is associated with the 18F-FDG PET/CT metabolic parameters, prognosis, and progression of esophageal cancer

Hua-Song Liu 1,†,#, Qiang Guo 1,†,#, Heng Yang 1,†,#, Min Zeng 1, Li-Qiang Xu 1, Qun-Xian Zhang 1, Hua Liu 1, Jia-Long Guo 1,*, Jun Zhang 1,*
PMCID: PMC13231007  PMID: 42245404

There was a mistake in Figure 9 as published. The representative images in Figures 9E,F of the control group cells were duplicated. Upon re-examination of the raw data, we found no issues with the control cells in the migration assay. However, the control cells in the invasion assay required re-counting. In this erratum, we re-counted the control cells in the invasion assay and observed a statistically significant difference between the experimental and control groups. This indicated that suppressing SPDL1 expression inhibits cell invasion, which was consistent with our previous results. The corrected Figure 9 and its caption appears below.

FIGURE 9.

Scientific figure showing effects of SPDL1 knockdown in EC109 cells, with six panels: A, B, and C display bar graphs and a line graph comparing SPDL1 expression and proliferation between si-NC and si-SPDL1; D shows immunofluorescence images and bar graph quantifying EdU-positive cells; E and F present representative images and quantification of cell migration and invasion, both reduced by SPDL1 knockdown; significant differences are marked by asterisks.

SPDL1 silencing inhibits ESCA cell growth and migration. (A,B) Establishment of ESCA cell model; (C,D) cell proliferation determined via Cell Counting Kit-8 and Edu methods and (E,F) cell migration and invasion determined using Transwell assays. ESCA, esophageal cancer; SPDL1, spindle apparatus coiled-coil protein 1.

The original article has been updated.

Footnotes

Edited and reviewed by: Marcelo R. S. Briones, Federal University of São Paulo, Brazil

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